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Welcome to Chiromed’s resource hub for Traumatic Brain Injuries (TBI)—including concussions, post-concussion syndrome, and whiplash-related head injuries. This category brings together evidence-informed guidance on TBI symptoms, concussion recovery, and integrative rehabilitation so you can navigate healing with clarity and confidence.

You’ll find easy-to-read articles on how TBIs happen—from sports collisions and falls to motor vehicle accidents—and what to watch for next: headaches, dizziness, neck pain, light and noise sensitivity, brain fog, and sleep disruption. We describe how the way your neck moves affects brain recovery, why exercises for balance and eye coordination are important, and how specific chiropractic and soft-tissue treatments can help lessen neck issues that often make symptoms last longer.

Our TBI content outlines a clear plan: gentle chiropractic adjustments to help joints move better and reduce pain signals; training for balance and eye coordination; breathing and posture techniques to help the nervous system; and simple nutrition advice—like staying hydrated, eating omega-3s, and choosing anti-inflammatory foods—to support brain recovery. We also cover safe return-to-learn and return-to-play progressions, red-flag warning signs, and when to seek imaging or specialist referral.

Whether you’re new to concussion care or managing lingering symptoms, this category is designed to help you:

Understand TBI timelines and realistic recovery expectations

Identify root contributors (neck, vision, vestibular, sleep, stress)

Build a stepwise, patient-centered plan that supports long-term brain health

Explore the latest posts below to learn how integrative chiropractic care fits into comprehensive post-concussion rehabilitation, and discover actionable steps you can start today. If you’re recovering from a recent head or neck injury, begin with our “TBI Basics” and “Neck & Concussion Connection” guides to create a safer, smarter path back to normal.

Beat TBIs and Body Toxicity with Chiropractic Care

Beat TBIs and Body Toxicity with Chiropractic Care

Healing from Within: How Traumatic Brain Injuries Create Body Toxicity and Integrative Care Supports Adult Recovery

Traumatic brain injuries, also known as TBIs, can abruptly alter a person’s life. For many adults, these injuries occur during a car crash on the way to work, a vicious hit in a weekend soccer game, or a fall at a construction site. These injuries do more than bruise the skull—they start a chain reaction of harm inside the body. This process creates a kind of “toxicity” that spreads from the brain to other organs, making recovery tough. But there’s hope. An integrative care approach, led by experts such as chiropractic nurse practitioners (CNPs), considers the whole person. It helps calm the body’s chaos, eases pain naturally, and builds strength for the long haul. Families and care teams also play a crucial role, providing emotional support and daily assistance. In this article, we’ll break down how TBIs cause this inner poison, why it matters for adults, and how team-based care can turn things around.

Imagine a 35-year-old office worker named Mark. He’s rear-ended in traffic, his head snaps back, and everything goes black for a moment. At first, it’s headaches and dizziness. Weeks later, gut issues and mood swings hit hard. The hidden side of TBI involves biochemical events that intensify over time. Research shows these effects can last weeks or years, raising risks for bigger problems like memory loss or even diseases like Alzheimer’s (Priester, 2025). But early, whole-body care changes the story. CNPs combine chiropractic adjustments with nursing expertise to reset the nervous system and combat inflammation. They guide adults like Mark back to work, play, and family life. This isn’t just medicine; it’s a roadmap for healing that honors the body’s own power.

For families, it’s personal. Spouses learn to spot warning signs, like when fatigue turns to frustration. Care teams coordinate visits, meals, and therapy sessions to ensure seamless care. Together, they tackle the toxicity head-on. As one study notes, addressing both the brain and body early can prevent long-term damage (Rauchman et al., 2023). Let’s dive into the science, simply explained, and see how recovery works in real life.

Understanding Traumatic Brain Injuries in Everyday Adult Life

Adults face TBIs more often than we think. In the U.S., over 2.8 million people seek emergency care each year, with motor vehicle accidents (MVAs) accounting for about 28%, falls at work for 20%, and sports-related injuries, such as those from football or boxing, making up another significant portion (Rauchman et al., 2023). A busy parent or factory worker can be out of work for months after a small slip or crash. Unlike children, adults often juggle jobs, bills, and family responsibilities, so recovery hits harder—lost wages, strained relationships, and endless doctor’s wait times.

A TBI starts with the primary injury: the direct hit. In an MVA, the brain slams against the skull, tearing blood vessels and nerves. Sports concussions come from rotational forces, twisting the brain like a wet towel. Workplace incidents, like dropping tools on the head, add blunt force. Right away, symptoms appear: confusion, nausea, and blurred vision. However, the real danger lies in the seconds that follow—the brain swells, pressure builds, and oxygen levels drop (Salehi et al., 2017).

Take Sarah, a 42-year-old soccer coach. A header in a pickup game leaves her with a mild concussion. She pushes through practices, but soon battles insomnia and irritability. Her family notices she’s “off.” This is common; mild TBIs affect 80% of cases, yet many adults ignore them, thinking it’s just a bump (Laskowitz & Grant, 2016). Men in their 30s and 40s, often in high-risk jobs or sports, make up the bulk. Women post-childbirth or in caregiving roles face extra stress, slowing healing.

Why does this matter? TBIs don’t stay in the head. They spark a body-wide alarm, releasing stress hormones that tax the heart and gut. Without quick care, simple tasks like driving become scary. But spotting it early helps. Doctors use CT scans for severe cases, but for mild ones, it’s a history and physical examination. Families step in here—tracking symptoms in a journal, urging rest. Workplaces can adapt with flexible hours or ergonomic fixes.

Symptom Questionnaire:

The positive news is that there are solutions available. Most adults recover well with support. One review found that 70% of patients return to normal within three months if treated holistically (Schimmel et al., 2017). That means blending rest, therapy, and family encouragement. For Mark from the intro, his wife joined therapy sessions, learning cues to de-escalate his frustration. It’s not just survival; it’s reclaiming life.

The Toxic Cascade: How TBIs Poison the Brain and Body

A TBI isn’t a one-and-done event. The initial impact, known as the primary injury, initiates a cascade of biochemical complications. This “cascade” turns the brain into a toxic zone, harming cells and spreading chaos to the gut, blood, and beyond. It’s like a fire that starts small but burns hot if unchecked. Understanding this helps adults and their teams fight back smarter.

Firstly, consider the initial impact. In an MVA, rapid deceleration shears axons—the brain’s wiring—like pulling threads from fabric. Sports-related impacts stretch tissue, while falling objects from work crush it. This releases danger signals, known as damage-associated molecular patterns (DAMPs), which alert the immune system (McKee & Lukens, 2016). Blood vessels break, starving cells of oxygen. Swelling, or edema, follows fast. There are two main types: cytotoxic, where cells suck up water like sponges due to pump failures, and vasogenic, where the blood-brain barrier (BBB) leaks like a busted dam, flooding tissue with proteins and fluid (Salehi et al., 2017). In adults, this raises skull pressure, squeezing the brain and risking more death. One study in mice showed edema peaking days after impact, mirroring human cases (Priester, 2025).

Now, the secondary storm—the real toxicity builder. It unfolds in phases: minutes, hours, days. Enter excitotoxicity. Damaged neurons release glutamate, the brain’s “go” signal, into the space. Normally, this excites cells briefly. However, in traumatic brain injury (TBI), it triggers a massive surge of glutamate. Glutamate overworks receptors, letting calcium rush in like floodwater. This calcium revs up destructive enzymes, which rip membranes and shred DNA. Cells swell, burst, and die in a chain reaction (Waters, n.d.). It’s why symptoms like seizures or coma are delayed. In car crashes, this “glutamate storm” spreads from impact zones, killing healthy neighbors (Rauchman et al., 2023). Adults in high-stress jobs often experience chronic fatigue, as their brains remain in overdrive.

Next, oxidative stress amps up the damage. The brain guzzles oxygen but has weak defenses. TBI sparks reactive oxygen species (ROS)—unstable molecules like superoxide or hydroxyl radicals—from busted mitochondria and fired-up immune cells. These ROS (reactive oxygen species) chew lipids in cell walls, creating toxic byproducts like 4-hydroxynonenal, which poison proteins and genes (Fesharaki-Zadeh, 2022). Iron from burst blood vessels fuels this process via Fenton reactions, generating more radicals. In sports concussions, repeated hits build ROS over time, explaining why pros face early Parkinson’s risks (Wu et al., 2022). One mouse study found that ROS stayed around for weeks after the infection, changing proteins and DNA in ways that are similar to the long-term symptoms of adults with persistent cognitive impairment (Priester, 2025).

Neuroinflammation piles on. Microglia, the brain’s guards, wake up and call in troops: monocytes via CCR2 signals and neutrophils, which release cytokines such as TNF-α and IL-1β (McKee & Lukens, 2016). This “fire” initially clears debris, but it then veers off course and attacks healthy tissue. In work injuries, chronic low-grade inflammation lingers, turning acute pain into a daily ache. Microglia also accumulate amyloid proteins, which serve as seeds for plaques in Alzheimer’s disease (Denniss & Barker, 2023). Cytokines breach the BBB, worsening leaks and edema. Adults report mood dips here—irritability from inflamed pathways mimicking depression.

Keep in mind the disruption of the gut-brain axis. The vagus nerve and microbes facilitate communication between the brain and gut. TBI shocks this link, slowing gut motility and poking holes in the intestinal wall—”leaky gut” (Faden et al., 2021). Bacteria enter the bloodstream, triggering sepsis or a body-wide inflammatory response. In MVAs, stress hormones like cortisol halt digestion, causing ulcers or symptoms similar to IBS (Heuer Fischer, P.A., n.d.). One study linked TBI-induced gut changes to worse brain swelling, as toxins circulate back via the blood (Cannon et al., 2023). For a construction worker, a post-fall condition means nausea on top of headaches, which can delay their return to the site.

These events interconnect: excitotoxicity generates ROS; inflammation widens the BBB cracks; gut leaks fuel the fire. The BBB, that tight shield of endothelial cells and astrocyte feet, frays from the action of matrix metalloproteinases (MMPs) and VEGF surges, allowing toxins to enter (Laskowitz & Grant, 2016a). Edema follows, compressing vessels and depriving cells of oxygen. In adults, this cascade hits harder—aging brains have less reserve, per one review (Salehi et al., 2017). However, is it possible to detect it at an early stage? Antioxidants, such as those in a new polymer, reduce ROS by 50% in mice, suggesting potential benefits in humans (Priester, 2025).

This toxicity isn’t abstract. For Sarah, the coach, it meant experiencing gut cramps and sidelining drills. Mark’s family adjusted meals to ease inflammation. Knowing the cascade empowers choice—enabling rest, consuming anti-inflammatory foods, and receiving targeted care. It’s the body’s cry for balance, and integrative pros listen.

Long-Term Risks: From Acute Toxicity to Lasting Brain Changes

If unchecked, TBI’s toxic wave doesn’t fade—it reshapes the brain. Weeks after the hit, waste like tau proteins piles up because the glymphatic system, the brain’s drain, clogs (Plog & Nedergaard, 2018). This mirrors the aging process or Alzheimer’s, where toxins spread, forming plaques. In adults, repeated sports hits can cause chronic traumatic encephalopathy (CTE)—mood swings, aggression, and dementia decades later (Priester, 2025).

Oxidative scars mutate genes; inflammation scars tissue with glial walls, blocking repair (Denniss & Barker, 2023). Gut leaks let endotoxins fuel chronic fatigue. One study tied early BBB breaks to poor outcomes years on (Laskowitz & Grant, 2016a). For work-hardened adults, this means early retirement and family strain. But mitigation works—lifestyle tweaks cut risks by 30% (Schimmel et al., 2017). It’s a wake-up: Act now, or pay later.

An Integrative Path to Recovery: The Role of Chiropractic Nurse Practitioners

Integrative care challenges the conventional understanding of TBI toxicity. It’s not just pills or scalpels—it’s a team that weaves chiropractic, nursing, nutrition, and therapy into one comprehensive plan. At the heart? Chiropractic nurse practitioners (CNPs). Trained in both fields, they identify spine-brain connections, adjust misalignments, and promote holistic healing. For adults post-MVA or concussion, this means less toxicity and more resilience.

Why chiropractic? The spine houses the nervous system; it conveys, constricts, and conveys signals. Adjustments realign the vertebrae, easing nerve pressure and resetting the “fight-or-flight” mode to a calm state (Sea Change Wellness Chiropractic, n.d.). One clinic notes it boosts cerebrospinal fluid (CSF) flow, the brain’s bath that clears toxins (Apex Chiropractic, n.d.). In workplace falls, this reduces headaches by 60%, according to patient reports (Northwest Florida Physicians Group, LLC, n.d.). CNPs add nursing layers by monitoring vitals, adjusting medications, and teaching self-care.

Dr. Alexander Jimenez, DC, APRN, FNP-BC, embodies this. At his El Paso clinic, he treats auto accident victims with spinal decompression and functional nutrition, targeting root causes like inflammation (Jimenez, n.d.a). “We restore normal functions after injuries without drugs,” he says, blending adjustments with omega-3s to douse ROS (Jimenez, n.d.b). His cases? A truck driver post-crash regained focus via neuropathy protocols; a golfer shook sports fog with vagus nerve stim via adjustments. Over 30 years, he’s seen integrative plans slash recovery time, empowering adults to ditch painkillers.

This approach hits all cascades. For excitotoxicity, gentle cranial work calms glutamate storms (Dr. Kal, n.d.). Oxidative stress? CNPs promote the uptake of antioxidants—such as berries and vitamin E—to neutralize ROS, a finding supported by mouse studies (Wu et al., 2022). Neuroinflammation can be alleviated with posture adjustments, thereby reducing cytokine triggers (Serenity Healthcare Partners, n.d.). Gut-brain? Probiotics and vagus-focused breathing mend leaks (Faden et al., 2021). BBB heals via better circulation from alignments.

Integrated therapies shine. Physical therapy helps rebuild balance, while CBT tames anxiety (Peixoto et al., 2025). Nutrition—anti-inflammatory diets—fuels repair (Serenity Healthcare Partners, n.d.). Emerging technologies, such as EMF stimulation in swine models, restore brain waves, hinting at potential human applications (Brazdzionis et al., 2023). CNPs coordinate, personalizing for a 50-year-old welder’s shifts or a mom’s school runs.

For Mark, CNP-led sessions mixed adjustments with family nutrition classes. Sarah added yoga for gut calm. Results? Sarah experienced faster clarity and fewer trips to the emergency room. Dr. Jimenez’s webinars stress this: “Functional medicine reverses imbalances—oxidative stress, gut dysbiosis—for true recovery” (Jimenez, n.d.b). It’s empowering, natural, and effective.

Supporting the Journey: Families and Care Teams in Adult TBI Recovery

Recovery isn’t solo. Families and care teams are the glue, turning plans into action. Spouses track moods, spotting toxicity flares like irritability from inflammation. Kids adapt games for dad’s fatigue; siblings share chores. This buffer cuts depression risks by 40% (Peixoto et al., 2025).

Care teams—CNPs, therapists, and docs—huddle weekly, adjusting for work stress or sports urges. Families attend education sessions to learn about edema signs or gut-friendly meal options. One family’s story: Post-concussion, they mapped “rest zones” at home, easing Mark’s load. Emotional tools, such as support groups, build resilience. As Dr. Jimenez notes, “Holistic care includes mind and spirit—families amplify healing” (Jimenez, n.d.a). It’s a shared victory.

Conclusion: Reclaiming Life After the Storm

TBIs from crashes, games, or jobs unleash a toxic cascade—excitotoxicity flooding cells, ROS scorching tissues, inflammation raging, and gut links breaking. For adults, it’s a body-wide battle, but integrative care, spearheaded by CNPs, counters it. Adjustments reset nerves, nutrition quells fires, and teams sustain hope. With families involved, recovery isn’t just possible—it’s transformative. As research evolves, from antioxidants to EMF, the path brightens. Adults like Mark and Sarah prove: Healing starts within but thrives together. Seek care early; your future self will thank you.

References

Apex Chiropractic. (n.d.). How chiropractic care can treat a traumatic brain injury. https://apexchiroco.com/updates/how-chiropractic-care-can-treat-a-traumatic-brain-injury/

Brazdzionis, J., Radwan, M. M., Thankam, F., Lal, M. R., Baron, D., Connett, D. A., Agrawal, D. K., & Miulli, D. E. (2023). A swine model of traumatic brain injury: Effects of neuronally generated electromagnetic fields and electromagnetic field stimulation on traumatic brain injury-related changes. Cureus, 15(11), e48992. https://doi.org/10.7759/cureus.48992

Cannon, A. R., Anderson, L. J., Galicia, K., Murray, M. G., Kamran, A. S., Li, X., Gonzalez, R. P., & Choudhry, M. A. (2023). Traumatic brain injury induced inflammation and GI motility dysfunction. Brain Sciences, 13(3), 414. https://doi.org/10.3390/brainsci13030414

Denniss, R. J., & Barker, L. A. (2023). Brain trauma and the secondary cascade in humans: Review of the potential role of vitamins in reparative processes and functional outcome. Neuropsychiatric Disease and Treatment, 19, 1693–1707. https://doi.org/10.2147/NDT.S415943

Dr. Kal. (n.d.). Chiropractic relief for accident head injuries. https://drkal.com/chiropractic-relief-for-accident-head-injuries/

Faden, A. I., Barrett, J. P., Stoica, B. A., & Henry, R. J. (2021). Bi-directional brain-systemic interactions and outcomes after TBI. Trends in Neurosciences, 44(5), 406–418. https://doi.org/10.1016/j.tins.2020.12.004

Fesharaki-Zadeh, A. (2022). Oxidative stress in traumatic brain injury. International Journal of Molecular Sciences, 23(21), 13000. https://doi.org/10.3390/ijms232113000

Heuer Fischer, P.A. (n.d.). TBI and gut health. https://www.heuerfischer.com/firm-overview/blog/tbi-and-gut-health/

Jimenez, A. (n.d.a.). Injury specialists. https://dralexjimenez.com/

Jimenez, A. (n.d.b.). Dr. Alexander Jimenez, DC, APRN, FNP-BC, IFMCP, CFMP, ATN ♛ – Injury Medical Clinic PA. https://www.linkedin.com/in/dralexjimenez/

Laskowitz, D., & Grant, G. (Eds.). (2016a). Blood–brain barrier pathophysiology following traumatic brain injury. In Translational research in traumatic brain injury. CRC Press/Taylor & Francis Group. https://www.ncbi.nlm.nih.gov/books/NBK326726/

Laskowitz, D., & Grant, G. (Eds.). (2016b). Neuroplasticity after traumatic brain injury. In Translational research in traumatic brain injury. CRC Press/Taylor & Francis Group. https://www.ncbi.nlm.nih.gov/books/NBK326735/

McKee, C. A., & Lukens, J. R. (2016). Emerging roles for the immune system in traumatic brain injury. Frontiers in Immunology, 7, 556. https://doi.org/10.3389/fimmu.2016.00556

Northwest Florida Physicians Group, LLC. (n.d.). Using chiropractic care to treat traumatic brain injuries. https://northwestfloridaphysiciansgroup.com/using-chiropractic-care-to-treat-traumatic-brain-injuries/

Peixoto, B., Cruz, M., & Ustares, V. (2025). Traumatic brain injury and neuropsychiatric consequences. Current Psychiatry Reports, 27(1), 1–12. https://doi.org/10.1007/s11920-024-01523-4

Plog, B. A., & Nedergaard, M. (2018). The glymphatic system in CNS health and disease. Neuron, 98(6), 1095–1118. (From rehabpub.com summary)

Priester, A. (2025, February 13). Traumatic brain injuries have toxic effects that last weeks after initial impact − an antioxidant material reduces this damage in mice. The Conversation. https://theconversation.com/traumatic-brain-injuries-have-toxic-effects-that-last-weeks-after-initial-impact-an-antioxidant-material-reduces-this-damage-in-mice-247655

Rauchman, S. H., Zubair, A., Jacob, B., Rauchman, D., Pinkhasov, A., & Placantonakis, D. G. (2023). Traumatic brain injury: Mechanisms, manifestations, and visual sequelae. Frontiers in Neuroscience, 17, 1090672. https://doi.org/10.3389/fnins.2023.1090672

Salehi, A., Zhang, J. H., & Obenaus, A. (2017). Response of the cerebral vasculature following traumatic brain injury. Journal of Cerebral Blood Flow & Metabolism, 37(10), 2320–2339. https://doi.org/10.1177/0271678X17701660

Schimmel, S. J., Acosta, S., & Lozano, D. (2017). Neuroinflammation in traumatic brain injury: A chronic response to an acute injury. Journal of Neurotrauma, 34(13), 2139–2147. https://doi.org/10.1089/neu.2016.4648

Sea Change Wellness Chiropractic. (n.d.). How chiropractic helps reset the nervous system after car crash trauma. https://seachangechiropractic.com/how-chiropractic-helps-reset-the-nervous-system-after-car-crash-trauma/

Serenity Healthcare Partners. (n.d.). How integrated therapies enhance recovery from traumatic brain injuries. https://www.serenityhealthcarepartners.com/how-integrated-therapies-enhance-recovery-from-traumatic-brain-injuries/

Waters, C. (n.d.). Excitotoxicity: A secondary injury in traumatic brain damage. https://www.charliewaterslaw.com/brain-injury/excitotoxicity-a-secondary-injury-in-traumatic-brain-damage/

Wu, A.-G., Yong, Y.-Y., Pan, Y.-R., Zhang, L., Wu, J.-M., Zhang, Y., Tang, Y., Wei, J., Yu, L., Law, B. Y.-K., Yu, C.-L., Liu, J., Lan, C., Xu, R.-X., Zhou, X.-G., & Qin, D.-L. (2022). Targeting Nrf2-mediated oxidative stress response in traumatic brain injury: Therapeutic perspectives of phytochemicals. International Journal of Molecular Sciences, 23(7), 3771. https://doi.org/10.3390/ijms23073771

Cognitive Impairment Overview With Traumatic Brain Injury


Learn how cognitive impairment relates to traumatic brain injury. Discover symptoms, diagnosis, and recovery strategies.

Introduction

One of the biggest health problems of our time is brain damage, which affects millions of individuals every year and has effects that endure long after the original injury. Over 30% of injury-related fatalities in the US include some kind of brain trauma, making traumatic brain injuries a leading cause of mortality and disability globally (Bailes & Borlongan, 2020). These wounds set off an intricate series of events that alter not only how the brain works but also how the body as a whole functions. missionlegalcenter Two separate stages of damage occur when a person gets a traumatic brain injury. The main harm occurs when external forces instantly induce mechanical damage to brain tissue at the point of contact. A secondary damage phase follows, when biochemical processes such as oxidative stress, inflammation, cell death, and other detrimental alterations cause the brain to gradually deteriorate over the course of days, weeks, and even months after the original trauma (Bailes & Borlongan, 2020). Healthcare professionals may create more effective treatment plans that address both short-term issues and long-term healing requirements by having a better understanding of these injury patterns. missionlegalcenter Cognitive impairment represents one of the most common and challenging consequences of traumatic brain injury. Problems with attention, memory, and executive functioning emerge as the primary neurocognitive consequences across all levels of injury severity (Cognitive Impairment Following Traumatic Brain Injury, 2002). These cognitive disruptions profoundly affect daily life, making it difficult for individuals to work, maintain relationships, manage household tasks, and participate fully in their communities. Because attention and memory serve as foundational cognitive abilities, their disruption can trigger additional problems with executive function, communication, and other complex mental processes (Cognitive Impairment Following Traumatic Brain Injury, 2002).pubmed.ncbi.nlm.nih

The connection between brain and body becomes especially important when considering traumatic brain injury recovery. The brain controls virtually every function in the human body through an intricate network of nerves and chemical signals. The central nervous system, comprising the brain and spinal cord, regulates awareness, movement, sensation, thought, speech, and memory (Anatomy and physiology of the nervous system, 2020). When injury disrupts these control centers, the effects ripple throughout the entire body, affecting muscles, bones, organs, and metabolic processes.cancer An integrative approach that combines chiropractic care with nurse practitioner oversight offers promising possibilities for individuals recovering from traumatic brain injuries. This collaborative model addresses the neurological, musculoskeletal, cognitive, emotional, and metabolic aspects of recovery. Chiropractic care focuses on restoring nervous system function through spinal adjustments, soft-tissue therapies, and targeted exercises, while nurse practitioners provide comprehensive medical management, cognitive support, and coordination of overall health needs. Together, these providers can create comprehensive treatment plans that support the brain’s natural healing processes and help patients regain function and improve their quality of life.

What is a Traumatic Brain Injury?

Traumatic brain injury refers to brain damage caused by an outside force that disrupts normal brain function. This external force can take many forms, including a forceful bump, blow, or jolt to the head or body, or an object penetrating the skull and damaging brain tissue (Traumatic Brain Injury, 2023). The injury occurs when the brain moves violently inside the skull or when an external object breaks through the skull barrier. Common causes include motor vehicle accidents, falls, sports injuries, violence, and blast exposures from explosions (Types of Traumatic Brain Injury, 2024).ninds.nih+1 Healthcare providers classify traumatic brain injuries according to their severity, which helps guide treatment decisions and predict outcomes. The three main categories include mild, moderate, and severe traumatic brain injury. Medical professionals use several measures to determine severity, including the Glasgow Coma Scale score, duration of loss of consciousness, length of post-traumatic amnesia, and results from brain imaging studies (Criteria used to classify TBI severity, 2012).ncbi.nlm.nih+1

  • Mild traumatic brain injury, often called a concussion, generally does not cause prolonged loss of consciousness. If unconsciousness occurs, it typically lasts less than thirty minutes. The Glasgow Coma Scale score ranges from thirteen to fifteen for mild injuries. Common symptoms include headaches, dizziness, confusion, nausea, vision problems, difficulty thinking clearly, balance issues, sleep disturbances, sensitivity to light and sound, problems with attention and concentration, fatigue, anxiety, irritability, and emotional changes (4 Types of Brain Injuries and 3 Levels of Severity, 2021). Memory loss associated with mild traumatic brain injury usually lasts less than twenty-four hours. Most people with mild injuries recover within a few days to weeks with appropriate rest and management (4 Types of Brain Injuries and 3 Levels of Severity, 2021).missionlegalcenter
  • Moderate traumatic brain injury involves unconsciousness lasting more than thirty minutes but less than twenty-four hours. The Glasgow Coma Scale score falls between nine and twelve. Individuals with moderate injuries experience all the symptoms associated with mild traumatic brain injury, plus additional concerning signs. These include headaches that worsen or do not improve, seizures or convulsions, numbness or weakness in the arms and legs, repeated vomiting, inability to wake from sleep, and slurred speech (4 Types of Brain Injuries and 3 Levels of Severity, 2021). Post-traumatic amnesia lasts more than one day but less than seven days. Brain imaging may show abnormalities such as bleeding, bruising, or swelling (Criteria used to classify TBI severity, 2012).ncbi.nlm.nih+1
  • Severe traumatic brain injury represents the most serious category, with loss of consciousness exceeding twenty-four hours. The Glasgow Coma Scale score ranges from three to eight. Post-traumatic amnesia persists for more than seven days. Individuals with severe injuries often require intensive medical care and extended rehabilitation. They may experience altered consciousness states, including coma, vegetative state, or minimally conscious state (Traumatic Brain Injury, 2023). Brain imaging typically reveals significant abnormalities including bleeding within the brain tissue, bleeding over the brain surface, bleeding in the brain’s ventricles, swelling, and tissue damage (Types of Traumatic Brain Injury, 2024).medschool.ucla+1

The type of traumatic brain injury also provides important classification information. Closed head injuries occur when the head experiences impact or rapid movement without skull penetration. Concussions, diffuse axonal injury, and contusions fall into this category. Diffuse axonal injury, one of the most common types, involves widespread damage to the brain’s white matter, which contains nerve fibers that facilitate communication between different brain regions. This type of injury commonly happens in auto accidents, falls, and sports-related trauma (Traumatic Brain Injury, 2023). Penetrating brain injuries occur when an object breaks through the skull and enters brain tissue, as seen with gunshot wounds or impalement injuries (Types of Traumatic Brain Injury, 2024).ninds.nih+1 Understanding whether an injury is primary or secondary helps guide treatment approaches. Primary traumatic brain injury refers to the immediate structural damage inside the brain resulting directly from the initial impact. This includes bruising, bleeding, and tearing of brain tissue and blood vessels. Secondary traumatic brain injury describes complications that develop after the initial trauma and cause additional damage to an already compromised brain. Secondary injury mechanisms include increased pressure inside the skull, progressive brain swelling, damage to blood vessels triggering stroke or seizures, and lack of oxygen related to blood pressure drops or breathing difficulties (Types of Traumatic Brain Injury, 2024).medschool.ucla

How TBI Causes Cognitive Impairment

Traumatic brain injury triggers a complex series of events that disrupt normal brain function and lead to cognitive impairment. Understanding these mechanisms enables healthcare providers to develop targeted interventions that support recovery and effectively manage symptoms.

  • The physical damage from traumatic brain injury affects brain structure and function in multiple ways. When the brain experiences trauma, nerve cells can be stretched, torn, or destroyed. The white matter tracts that connect different brain regions become damaged, disrupting the communication networks essential for coordinated brain function. Diffuse axonal injury particularly affects these communication pathways, as the nerve fibers that transmit signals between brain cells break down and lose their ability to conduct information efficiently (Traumatic Brain Injury, 2023).ninds.nih
  • Bleeding within the brain creates additional problems. When blood vessels rupture, blood accumulates in spaces where it does not belong, creating pressure that compresses surrounding brain tissue. This compression damages cells both directly through physical pressure and indirectly by reducing blood flow to affected areas. Swelling further compounds these problems, as increased fluid within the rigid skull creates mounting pressure that can damage brain tissue and reduce oxygen delivery (Types of Traumatic Brain Injury, 2024).medschool.ucla

At the cellular level, traumatic brain injury initiates harmful biochemical cascades. Cell membranes become disrupted, allowing excessive calcium and sodium to enter neurons. This triggers a series of destructive processes including activation of enzymes that break down cellular components, production of free radicals that damage cell structures, mitochondrial dysfunction that impairs energy production, and release of inflammatory molecules that promote further injury (Bailes & Borlongan, 2020). These processes can continue for days, weeks, or even months after the initial injury, explaining why symptoms sometimes worsen or new problems emerge well after the traumatic event.missionlegalcenter

  • Inflammation plays a particularly important role in post-traumatic brain injury cognitive impairment. Within seconds after trauma, inflammatory responses activate in the brain. The blood-brain barrier, which normally protects the brain from harmful substances in the bloodstream, becomes damaged and allows inflammatory cells and molecules to enter brain tissue. While some inflammation helps with healing and clearing damaged tissue, excessive or prolonged inflammation damages healthy brain cells and interferes with recovery. Inflammatory molecules affect neurotransmitter systems, disrupt nerve signaling, and impair the formation of new neural connections needed for cognitive recovery (Mesenchymal stem cell therapy alleviates the neuroinflammation, 2020).medicine.washu

Different brain regions show varying vulnerability to traumatic injury, which explains the specific cognitive impairments that develop. The frontal lobes, responsible for executive functions such as planning, decision-making, impulse control, and working memory, are particularly susceptible to damage from trauma. The temporal lobes, involved in memory formation and language processing, also commonly sustain injury. Damage to the hippocampus, a structure critical for forming new memories, explains why memory problems rank among the most frequent cognitive complaints after traumatic brain injury (Cognitive Problems After Traumatic Brain Injury, n.d.).uwmsktc.washington

  • Attention and concentration problems emerge as foundational deficits following traumatic brain injury. Individuals may struggle to focus, pay attention to relevant information while filtering out distractions, or attend to more than one task at a time. This leads to restlessness, easy distractibility, difficulty finishing projects, problems carrying on conversations, and trouble sitting still for extended periods. Because attention skills serve as building blocks for higher-level cognitive abilities, people with attention problems often develop additional difficulties with memory, reasoning, and problem-solving (Cognitive Problems After Traumatic Brain Injury, n.d.).uwmsktc.washington
  • Processing speed commonly slows after brain injury. Individuals take longer to understand what others are saying, need more time to follow directions, struggle to keep up with television shows or movies, require additional time to read and comprehend written material, and show delayed reactions to stimuli. This slowed processing affects everyday activities and can make tasks that were once automatic feel laborious and exhausting. The reduced reaction time poses particular concerns for activities requiring quick responses, such as driving (Cognitive Problems After Traumatic Brain Injury, n.d.).uwmsktc.washington
  • Memory impairments manifest in various ways after traumatic brain injury. Short-term memory problems make it difficult to hold information in mind temporarily, such as remembering a phone number long enough to dial it or recalling items on a shopping list. Long-term memory difficulties affect the ability to store and retrieve information over extended periods. People may struggle to remember recent events, learn new information, or recall facts and procedures they previously knew well. Working memory, which involves holding and manipulating information simultaneously, becomes compromised, affecting complex cognitive tasks like mental arithmetic, following multi-step directions, and reasoning (Cognitive Impairment Following Traumatic Brain Injury, 2002).pubmed.ncbi.nlm.nih
  • Executive function deficits represent another hallmark of traumatic brain injury cognitive impairment. Executive functions include the mental processes that help people plan activities, organize information, initiate tasks, monitor performance, shift between tasks flexibly, solve problems, make decisions, and control impulses. When these abilities become impaired, individuals struggle with goal-directed behavior, adapting to new situations, regulating emotions, and functioning independently in daily life (Cognitive Problems After Traumatic Brain Injury, n.d.).uwmsktc.washington

The Brain-Body Connection

The relationship between the brain and body represents one of the most fundamental aspects of human physiology. This intricate connection enables all body functions, from voluntary movements to unconscious processes that sustain life. Understanding this connection becomes especially important when considering how traumatic brain injury affects not just cognitive abilities but overall physical health and function.

  • The central nervous system serves as the command center for the entire body. Made up of the brain and spinal cord, this system controls awareness, movements, sensations, thoughts, speech, and the five senses of seeing, hearing, feeling, tasting, and smelling (Central nervous system function, 2025). The brain manages most body functions by processing information from sensory receptors throughout the body and sending out instructions through an extensive network of nerves. The spinal cord acts as an extension of the brain, carrying messages between the brain and peripheral nerves that reach every part of the body (Central nervous system function, 2025).healthdirect
  • The peripheral nervous system complements the central nervous system by connecting the brain and spinal cord to the rest of the body. This network of nerves and ganglia sends signals to and receives signals from the central nervous system, enabling two-way communication between the brain and body tissues. The peripheral nervous system divides into the somatic nervous system, which controls voluntary movements like walking and grasping objects, and the autonomic nervous system, which manages involuntary functions that the body performs automatically, such as breathing, heartbeat, digestion, and blood pressure regulation (Anatomy and physiology of the nervous system, 2020).cancer
  • The autonomic nervous system further separates into two complementary branches that maintain balance in body functions. The sympathetic nervous system prepares the body for situations requiring strength, heightened awareness, or rapid response, commonly known as the fight-or-flight response. Activation of this system increases heart rate, elevates blood pressure, speeds breathing, dilates pupils, and increases metabolic rate. The parasympathetic nervous system creates opposite effects, returning heart rate and breathing to normal, constricting pupils, and slowing metabolism to conserve energy and promote rest and recovery (Anatomy and physiology of the nervous system, 2020).cancer
  • Research demonstrates that the brain and body maintain constant, bidirectional communication through multiple pathways. Recent studies show that parts of the brain area controlling movement connect directly to networks involved in thinking, planning, and control of involuntary body functions such as blood pressure and heartbeat. This literal linkage of body and mind in brain structure helps explain phenomena like why anxiety makes people pace, why vagus nerve stimulation can alleviate depression, and why regular exercise improves mental outlook (Mind-body connection is built into brain, 2023).medicine.washu
  • The vagus nerve exemplifies this brain-body connection. This cranial nerve carries signals between the brain and internal organs, providing information about organ function and regulating processes like digestion and heart rate. Signals traveling through the vagus nerve are coded independently by specialized neurons, allowing the brain to discriminate precisely among various body signals and respond appropriately. This sophisticated communication system enables the brain to monitor and adjust organ function continuously based on changing body needs and environmental demands (Revealing Communications Between Brain and Body, 2022).medicine.yale
  • Blood flow represents another critical aspect of brain-body connection. The brain, despite constituting only about two percent of total body mass, consumes over twenty percent of the body’s glucose-derived energy. Continuous glucose metabolism supports neuronal signaling, as adenosine triphosphate, the cell’s energy currency, powers action potentials, maintains ionic gradients, and supports synaptic transmission. Because the brain cannot synthesize or store glucose independently, it depends entirely on glucose from dietary intake and blood circulation. Any disruption to blood flow or energy metabolism can significantly impair brain function (Metabolic hormones mediate cognition, 2009).sciencedirect
  • The musculoskeletal system connects intimately with brain function through sensory feedback and motor control. Muscles contain specialized receptors that constantly send information to the brain about body position, movement, and force. This proprioceptive feedback allows the brain to coordinate movement, maintain posture, and adjust to environmental demands. The brain processes this information and sends motor commands back to muscles, enabling precise, coordinated movement. When traumatic brain injury disrupts these communication pathways, both sensory perception and motor control become impaired (Nervous System Function, 2024).clevelandclinic
  • Hormonal systems provide another dimension of brain-body connection. The hypothalamus and pituitary gland, located deep within the brain, regulate hormonal signals that control growth, metabolism, reproduction, stress response, and many other functions. These structures form a feedback loop, with the hypothalamus releasing hormones that signal the pituitary gland, which then distributes hormones to various body systems including the adrenal glands, thyroid, reproductive organs, skin, bone, and muscle. This hormonal regulation affects mood, memory, metabolism, muscle mass, energy levels, stress response, and reproductive function (Neuroendocrine Disturbances Following TBI, 2023).biausa
  • The immune system also maintains constant communication with the brain. Immune cells and inflammatory molecules can cross from the bloodstream into brain tissue, particularly when the blood-brain barrier becomes damaged following injury. The brain, in turn, can influence immune function through neural and hormonal signals. This bidirectional communication becomes particularly important following traumatic brain injury, when both local brain inflammation and systemic immune responses affect recovery and long-term outcomes (Multiorgan Dysfunction After Severe TBI, 2021).pmc.ncbi.nlm.nih

Causes and Symptoms of Cognitive Impairment

Cognitive impairment following traumatic brain injury arises from multiple interrelated causes that affect brain structure and function. Understanding these causes enables healthcare providers to identify risk factors, develop effective prevention strategies, and tailor targeted treatment approaches.

  • The primary cause of cognitive impairment stems from direct damage to brain tissue at the moment of injury. When the brain experiences sudden acceleration, deceleration, or rotational forces, nerve cells stretch and tear, blood vessels rupture, and tissue bruises. The specific location and extent of damage determine which cognitive functions become impaired. Injuries to the frontal lobes typically affect executive functions, attention, and working memory. Damage to the temporal lobe disrupts memory formation and language processing. Parietal lobe injuries interfere with sensory processing and spatial awareness, while occipital lobe damage affects visual processing (Traumatic Brain Injury, 2023).ninds.nih
  • Secondary injury mechanisms compound the initial damage. Swelling increases pressure within the rigid skull, compressing brain tissue and reducing blood flow. Bleeding creates masses that displace normal brain structures and increase intracranial pressure. Chemical imbalances develop as damaged cells release excessive amounts of neurotransmitters, particularly glutamate, which overstimulates neighboring neurons and triggers cell death. Free radicals produced during cellular metabolism damage cell membranes and DNA. Mitochondrial dysfunction impairs energy production, leaving neurons unable to maintain normal function. These secondary processes continue for days to weeks after the initial injury, explaining why cognitive symptoms may worsen or emerge gradually (Bailes & Borlongan, 2020).missionlegalcenter
  • Inflammation represents a major contributor to cognitive impairment following traumatic brain injury. The inflammatory response activates within seconds after trauma and can persist for months or even years. While acute inflammation helps remove damaged tissue and initiate healing, chronic inflammation damages healthy neurons and interferes with recovery. Inflammatory molecules disrupt neurotransmitter systems, impair synaptic plasticity, reduce the production of growth factors needed for neural repair, and contribute to the ongoing death of brain cells. This persistent inflammation particularly affects cognitive functions requiring complex neural networks and plasticity, such as learning, memory consolidation, and executive function (Mesenchymal stem cell therapy alleviates the neuroinflammation, 2020).medicine.washu
  • Disrupted blood flow contributes to cognitive impairment by reducing oxygen and nutrient delivery to brain tissue. Traumatic brain injury can damage blood vessels directly, alter blood pressure regulation, and trigger vasospasm where blood vessels constrict excessively. The brain requires constant, abundant blood supply to meet its high metabolic demands. Even brief or partial reductions in blood flow can impair neural function and contribute to cell death. Chronic reductions in cerebral blood flow may explain some persistent cognitive deficits that remain long after the initial injury (Long-term Consequences of TBI in Bone, 2018).pmc.ncbi.nlm.nih
  • Hormonal disruptions following traumatic brain injury affect cognition through multiple pathways. The hypothalamus and pituitary gland, structures that regulate hormonal systems, are particularly vulnerable to traumatic injury due to their location and delicate structure. Damage to these areas causes hypopituitarism, a condition where insufficient hormone production affects growth, metabolism, stress response, and reproduction. Growth hormone deficiency, thyroid hormone deficiency, and sex hormone deficiencies all contribute to cognitive impairment, affecting memory, attention, processing speed, and executive function (Neuroendocrine Disturbances Following TBI, 2023).biausa

The symptoms of cognitive impairment following traumatic brain injury vary widely depending on injury severity, location, and individual factors. Attention and concentration problems rank among the most common complaints. Individuals struggle to focus on tasks, become easily distracted by environmental stimuli, have difficulty filtering out irrelevant information, and cannot maintain attention for extended periods. These problems make it challenging to follow conversations, complete work tasks, read for comprehension, or perform activities requiring sustained mental effort (Cognitive Problems After Traumatic Brain Injury, n.d.).uwmsktc.washington

  • Memory impairments manifest in various ways. Short-term memory problems make it difficult to remember recent events, conversations, or instructions. People may repeatedly ask the same questions, forget appointments, or lose track of items. Long-term memory difficulties affect the ability to recall past events, previously learned information, or familiar procedures. Working memory deficits interfere with tasks requiring simultaneous information holding and manipulation, such as mental calculations, following multi-step directions, or reasoning through problems (Cognitive Impairment Following Traumatic Brain Injury, 2002).pubmed.ncbi.nlm.nih
  • Processing speed reductions cause delays in understanding and responding to information. Individuals take longer to comprehend spoken or written language, need extra time to formulate responses, show slowed reaction times, and struggle to keep pace in conversations or fast-moving situations. This slowed processing affects virtually all cognitive tasks and creates frustration when individuals recognize their difficulties but cannot overcome them through effort alone (Cognitive Problems After Traumatic Brain Injury, n.d.).uwmsktc.washington
  • Executive function deficits create problems with higher-order cognitive processes. People struggle with planning and organizing activities, initiating tasks without prompting, maintaining focus on long-term goals, shifting flexibly between tasks or mental sets, monitoring their own performance, solving novel problems, making sound decisions, and controlling impulses. These difficulties severely impact independence, as they interfere with managing finances, maintaining employment, keeping appointments, completing household tasks, and regulating behavior in social situations (Cognitive Problems After Traumatic Brain Injury, n.d.).uwmsktc.washington
  • Communication problems often accompany cognitive impairment. Individuals may have difficulty finding the right words, organizing their thoughts coherently, following complex conversations, understanding nonliteral language like sarcasm or idioms, interpreting social cues, or maintaining appropriate topics in conversation. These challenges affect relationships and social participation, contributing to isolation and reduced quality of life (Cognitive Impairment Following Traumatic Brain Injury, 2002).pubmed.ncbi.nlm.nih
  • Learning difficulties emerge when cognitive impairment affects the ability to acquire new information or skills. People need more repetition to learn new material, struggle to transfer learned skills to new situations, have difficulty recognizing patterns, and cannot efficiently organize information for storage and retrieval. These learning problems affect vocational rehabilitation, academic pursuits, and adaptation to life changes necessitated by the injury (Cognitive Impairment Following Traumatic Brain Injury, 2002).pubmed.ncbi.nlm.nih

Effects on Musculoskeletal and Neurological Systems

Traumatic brain injury creates widespread effects throughout the musculoskeletal and neurological systems, affecting movement, coordination, sensation, and physical integrity. These effects arise from both direct injury to neural structures that control these systems and secondary changes that develop over time. The musculoskeletal system experiences significant impacts following traumatic brain injury through multiple mechanisms. Spasticity, characterized by increased muscle tone and involuntary muscle contractions, develops in a substantial proportion of individuals with moderate to severe traumatic brain injury. The degree of spasticity varies from mild muscle stiffness to severe, painful, uncontrollable muscle spasms. Affected muscles may resist passive stretching, contract involuntarily, and develop shortened resting length over time. Spasticity interferes with movement, positioning, comfort, and functional activities. It can lead to joint contractures, pain, skin breakdown, and difficulty with daily care (TBI-Induced Spasticity, 2015).ncbi.nlm.nih

  • Muscle weakness and paralysis occur when traumatic brain injury damages motor cortex areas or descending motor pathways that transmit movement commands from brain to muscles. The pattern and severity of weakness depend on injury location. Hemiparesis, weakness affecting one side of the body, develops when injury occurs to motor areas in one brain hemisphere. Quadriparesis involves weakness in all four limbs. Even mild weakness significantly impacts function, affecting walking, reaching, grasping, and other essential movements. Muscle atrophy, or wasting, develops over time when muscles cannot be used normally due to weakness or inactivity (Physical effects of brain injury, n.d.).headway
  • Balance and coordination problems represent common musculoskeletal consequences of traumatic brain injury. Damage to the cerebellum, a brain structure that coordinates movement, causes ataxia characterized by unsteady gait, difficulty with fine motor tasks, tremor during purposeful movements, and impaired ability to judge distances. Balance problems also arise from vestibular system damage, proprioceptive deficits, visual processing impairments, and motor control difficulties. These balance and coordination deficits increase fall risk, limit mobility, and reduce independence in daily activities (Physical effects of brain injury, n.d.).headway
  • Post-traumatic seizures develop in some individuals following traumatic brain injury, representing neurological system dysfunction. Seizures can occur immediately after injury, within the first week, or months to years later. They result from abnormal electrical activity in damaged brain tissue. The risk increases with injury severity, presence of bleeding in the brain, skull fractures, and penetrating injuries. Seizures interfere with daily activities, increase injury risk, and may worsen cognitive impairment if not well controlled (Traumatic Brain Injury, 2023).ninds.nih
  • Sensory disturbances commonly accompany traumatic brain injury. Individuals may experience numbness, tingling, burning sensations, or altered temperature perception. Pain syndromes develop, including headaches, neck pain, and widespread body pain. These sensory changes result from damage to sensory processing areas in the brain, peripheral nerves, or spinal structures often injured concurrently with traumatic brain injury. Chronic pain significantly affects quality of life, mood, sleep, and rehabilitation participation (Pain and Traumatic Brain Injury, 2024).health
  • Vestibular dysfunction affects up to fifty percent of traumatic brain injury patients at five years post-injury. The vestibular system, which controls balance and spatial orientation, can be damaged at the peripheral level in the inner ear, at the central level in the brain, or both. Common vestibular diagnoses following traumatic brain injury include benign paroxysmal positional vertigo, where calcium crystals in the inner ear become displaced causing brief spinning sensations with position changes; acute unilateral peripheral vestibular loss, where one inner ear loses function; and migraine-associated vertigo. Vestibular dysfunction causes dizziness, vertigo, imbalance, nausea, and difficulty with activities requiring head movement. Interestingly, many individuals with objective vestibular dysfunction do not report symptoms, likely because traumatic brain injury affects perceptual mechanisms (Vestibular dysfunction in acute TBI, 2019).pmc.ncbi.nlm.nih
  • Vision and eye movement problems affect up to ninety percent of traumatic brain injury patients. These problems include difficulty tracking moving objects smoothly, impaired ability to shift gaze rapidly between targets, reduced convergence ability needed for near vision tasks, double vision from misalignment of the eyes, difficulty focusing, reduced visual field, and light sensitivity. These visual disturbances result from damage to cranial nerves that control eye muscles, brain areas that process visual information, or brain regions that coordinate eye movements. Visual dysfunction significantly impacts reading, driving, balance, and participation in rehabilitation activities (Eye Movement Problems After Brain Injury, 2021).optometrists+1
  • The skeletal system experiences long-term consequences from traumatic brain injury that are less obvious but clinically significant. Research shows that traumatic brain injury patients have increased risk of osteopenia and osteoporosis, conditions characterized by reduced bone mineral density and increased fracture risk. Bone loss occurs through multiple mechanisms, including reduced physical activity, hormonal disruptions affecting bone metabolism, vitamin D deficiency, inflammation, and altered bone formation and resorption signaling. Adults with traumatic brain injury show accelerated bone mineral density loss in the femur, particularly within the first year after injury. This increased skeletal fragility raises concern for future fractures that could complicate recovery and independence (Long-term Consequences of TBI in Bone, 2018).pmc.ncbi.nlm.nih
    • Heterotopic ossification, the formation of bone in soft tissues where bone should not normally exist, develops in some traumatic brain injury patients. This condition commonly affects muscles and soft tissues around major joints, particularly the hips, knees, elbows, and shoulders. Heterotopic ossification causes pain, limits joint range of motion, and interferes with positioning and movement. The mechanisms involve altered signaling from the injured brain that activates bone-forming cells in abnormal locations, increased inflammation, and changes in local blood flow (Long-term Consequences of TBI in Bone, 2018).pmc.ncbi.nlm.nih

Effects on Vital Organs

Traumatic brain injury extends its impact beyond the brain to affect vital organs throughout the body. This multiorgan dysfunction occurs through autonomic nervous system disruption, inflammatory mediators, hormonal changes, and metabolic alterations that the injured brain cannot properly regulate.

  • The cardiovascular system experiences significant effects following traumatic brain injury. Severe injuries trigger massive catecholamine release and autonomic nervous system activation, leading to elevated heart rate, increased blood pressure, and altered heart rhythm. While these changes may initially help maintain blood flow to the injured brain, they can become harmful if excessive or prolonged. Cardiac complications include neurogenic stress cardiomyopathy, where the heart muscle weakens temporarily; cardiac arrhythmias; and increased myocardial oxygen demand that can trigger ischemia in vulnerable individuals. Blood pressure dysregulation complicates management, as both very high and very low blood pressure can worsen brain injury outcomes (Multiorgan Dysfunction After Severe TBI, 2021).pmc.ncbi.nlm.nih
  • The pulmonary system suffers frequent complications after traumatic brain injury. Acute lung injury develops in many patients with severe brain trauma due to neurogenic pulmonary edema, where fluid accumulates in the lungs from autonomic nervous system dysfunction and altered blood vessel permeability. Pneumonia occurs frequently due to impaired ability to protect the airway, reduced cough effectiveness, and prolonged mechanical ventilation when required. Acute respiratory distress syndrome, a severe form of lung injury, can develop. These pulmonary complications reduce oxygen delivery to the injured brain and other organs, potentially worsening outcomes (Multiorgan Dysfunction After Severe TBI, 2021).pmc.ncbi.nlm.nih
  • The gastrointestinal system demonstrates vulnerability to traumatic brain injury effects. Autonomic nervous system disruption alters gut motility, reduces blood flow to intestinal tissues, and changes the gut microbiome composition. These changes increase intestinal permeability, potentially allowing bacteria and bacterial products to enter the bloodstream. Stress ulcers develop in the stomach and duodenum from reduced mucosal blood flow and altered protective mechanisms. Feeding intolerance complicates nutritional support. Gastrointestinal complications affect nutrient absorption, contribute to systemic inflammation, and may influence brain recovery (Multiorgan Dysfunction After Severe TBI, 2021).pmc.ncbi.nlm.nih
  • Kidney function becomes impaired in many traumatic brain injury patients through multiple mechanisms. Sympathetic nervous system activation reduces blood flow to the kidneys, decreasing glomerular filtration. Inflammatory mediators released from the injured brain affect kidney cells directly. Acute kidney injury develops in a significant proportion of patients with severe traumatic brain injury, potentially requiring dialysis and affecting long-term kidney function. Impaired kidney function complicates medication dosing, fluid management, and elimination of metabolic waste products (Multiorgan Dysfunction After Severe TBI, 2021).pmc.ncbi.nlm.nih
  • The liver, which synthesizes proteins and lipids crucial for brain recovery, experiences altered function following traumatic brain injury. Inflammatory signals affect hepatic protein synthesis, lipid metabolism, and glucose production. The liver may become a source of inflammatory mediators that worsen brain injury. Liver dysfunction affects drug metabolism, coagulation factor production, and nutritional status. Recent research suggests the liver plays a crucial role in traumatic brain injury pathogenesis through its metabolic and inflammatory functions (Traumatic brain injury from a peripheral axis perspective, 2025).sciencedirect
  • Metabolic and endocrine systems show widespread dysfunction after traumatic brain injury. The hypothalamic-pituitary axis, which regulates hormonal systems, commonly sustains damage. This results in deficiencies of growth hormone, thyroid hormone, adrenal hormones, and sex hormones. Growth hormone deficiency contributes to muscle wasting, bone loss, fatigue, and cognitive impairment. Thyroid hormone deficiency slows metabolism, affects mood and cognition, and impairs recovery. Adrenal insufficiency compromises stress response and blood pressure regulation. Sex hormone deficiencies affect mood, energy, muscle mass, and bone density. These hormonal disturbances can develop acutely or emerge months to years after injury, emphasizing the need for ongoing monitoring (Neuroendocrine Disturbances Following TBI, 2023).biausa
  • Blood sugar regulation becomes disrupted following traumatic brain injury, with both hyperglycemia and hypoglycemia occurring. The injured brain has altered glucose metabolism and increased metabolic demands. Insulin resistance can develop, affecting cellular energy metabolism throughout the body. These metabolic changes complicate nutritional management and may affect recovery outcomes. Evidence suggests that metabolic dysregulation contributes to cognitive impairment, as insulin and other metabolic hormones influence neuroplasticity and synaptic function (Metabolic hormones mediate cognition, 2009).sciencedirect

A TBI Symptom Questionnaire Example:

Detailed History and Questioning by Providers

Comprehensive assessment through detailed history-taking and systematic questioning forms the foundation of effective traumatic brain injury care. Both chiropractors and nurse practitioners use specific strategies to uncover cognitive impairment and identify the full scope of injury-related problems. A thorough history begins with understanding the mechanism of injury. Providers need detailed information about how the traumatic event occurred, including the forces involved, direction of impact, presence of acceleration or deceleration, rotational forces, and any loss of consciousness. This information helps predict injury patterns and potential complications. For example, motor vehicle accidents often cause both brain injury and cervical spine trauma, blast injuries affect multiple organ systems, and falls in older adults carry high risk for bleeding complications (Survey of chiropractic clinicians on MTBI, 2018).pmc.ncbi.nlm.nih

  • Timeline documentation provides essential context for symptom development. Providers should ask when symptoms first appeared, whether they emerged immediately after injury or developed gradually, how symptoms have changed over time, and whether any factors make symptoms better or worse. Some traumatic brain injury symptoms appear immediately, while others develop days, weeks, or months later. This temporal pattern helps distinguish primary injury effects from secondary complications and guides treatment planning (Survey of chiropractic clinicians on MTBI, 2018).pmc.ncbi.nlm.nih Cognitive symptoms require detailed exploration through specific questioning. Providers should systematically assess attention and concentration by asking about distractibility, ability to complete tasks, difficulty maintaining focus during conversations or activities, and need for frequent breaks. Memory problems should be explored across multiple domains, including difficulty remembering recent events, appointments, or conversations; problems with learning new information; struggles with recalling previously known facts or procedures; and concerns expressed by family members about changes in memory. Executive function difficulties often manifest as problems with planning, organizing, initiating tasks, managing time, making decisions, solving problems, and regulating emotions (Cognitive Problems After Traumatic Brain Injury, n.d.).uwmsktc.washington
  • Musculoskeletal symptoms deserve thorough investigation because they often accompany cognitive impairment and affect rehabilitation. Providers should ask about neck pain, back pain, headaches, dizziness, balance problems, muscle weakness, numbness or tingling, muscle stiffness or spasms, and changes in coordination or movement. The cervical spine frequently sustains injury concurrently with traumatic brain injury, and cervical dysfunction can contribute to headaches, dizziness, and cognitive symptoms through its effects on blood flow and proprioceptive input (Chiropractic Management of Post Traumatic Vertigo, 2004).pmc.ncbi.nlm.nih Vestibular symptoms require specific questioning because they are common but often underreported. Providers should directly ask about dizziness, vertigo, lightheadedness, imbalance, motion sensitivity, visual disturbances with movement, and situations that provoke symptoms. Many traumatic brain injury patients have vestibular dysfunction but do not report symptoms spontaneously, possibly because brain injury affects symptom perception. Direct questioning reveals these problems that might otherwise remain unidentified (Vestibular dysfunction in acute TBI, 2019).pmc.ncbi.nlm.nih  
  • Visual symptoms affect the majority of traumatic brain injury patients and significantly impact function. Providers should systematically assess blurred vision, double vision, difficulty focusing, eye strain, light sensitivity, problems tracking moving objects, difficulty with reading, visual field deficits, and eye misalignment. Because visual dysfunction contributes to balance problems, reading difficulties, and participation limitations, thorough visual assessment guides appropriate referrals and treatment planning (Eye Movement Problems After Brain Injury, 2021).optometrists Sleep disturbances occur in thirty to seventy percent of traumatic brain injury patients and affect recovery. Providers should ask about difficulty falling asleep, frequent nighttime awakenings, early morning awakening, excessive daytime sleepiness, prolonged sleep need, nightmares, and changes in sleep schedule or quality. Sleep disruption worsens cognitive function, mood, pain perception, and overall recovery. Identifying sleep problems allows targeted interventions that may improve multiple outcome domains (Sleep Disorders After Brain Injury, 2025).practicalneurology
  • Mood and emotional symptoms commonly develop after traumatic brain injury and require sensitive, direct questioning. Depression affects forty to sixty percent of individuals with moderate to severe traumatic brain injury. Symptoms include persistent sadness, loss of interest in previously enjoyed activities, feelings of hopelessness, changes in appetite, sleep disturbances, fatigue, difficulty concentrating, and suicidal thoughts. Anxiety disorders affect eleven to seventy percent of traumatic brain injury patients, with symptoms including excessive worry, restlessness, tension, hypervigilance, and panic attacks. Emotional dysregulation may manifest as irritability, anger outbursts, emotional lability, or apathy (Mood Disorders Following TBI, 2025).practicalneurology

Functional impacts should be thoroughly explored to understand how symptoms affect daily life. Providers should ask about changes in work or school performance, difficulty managing household tasks, problems maintaining relationships, challenges with self-care activities, driving limitations, and overall quality of life. Understanding functional limitations helps prioritize treatment goals and measure progress over time. Family member or caregiver input provides valuable perspective on functional changes that patients may not fully recognize (Strategies Nurses Use when Caring for Patients with TBI, 2019).pmc.ncbi.nlm.nih Inquiry about significant others’ observations proves particularly valuable, as cognitive impairment can affect self-awareness. Studies show that seventy to eighty-eight percent of healthcare providers inquire about family members’ observations of cognitive changes. Family members often notice personality changes, memory problems, emotional shifts, and functional declines that patients minimize or do not recognize (Survey of chiropractic clinicians on MTBI, 2018).pmc.ncbi.nlm.nih

Associated Symptoms from TBI

Beyond cognitive impairment, traumatic brain injury produces a constellation of associated symptoms that significantly affect quality of life and recovery. Understanding these symptoms helps providers develop comprehensive treatment approaches and set realistic expectations for recovery.

  • Fatigue represents one of the most common and debilitating symptoms after traumatic brain injury. Research indicates that as many as ninety-eight percent of people who have experienced traumatic brain injury have some form of fatigue. This fatigue differs from normal tiredness in that it does not improve adequately with rest, appears disproportionate to activity level, and significantly limits function. Physical fatigue manifests as muscle weakness, reduced endurance, and increased need for rest. Mental fatigue involves reduced ability to sustain cognitive effort, difficulty concentrating as the day progresses, and overwhelming sense of mental exhaustion. Fatigue worsens other symptoms, including pain, cognitive problems, and mood disturbances (Fatigue After Brain Injury, 2021).biausa
  • Headaches affect up to eighty percent of traumatic brain injury survivors and may persist for months or years. Post-traumatic headaches take various forms, including tension-type headaches characterized by band-like pressure, migraine-type headaches with throbbing pain and associated symptoms, cervicogenic headaches originating from neck dysfunction, and neuralgic headaches involving specific nerve distributions. Headaches interfere with concentration, sleep, mood, and participation in rehabilitation activities. The mechanisms involve inflammation, altered pain processing, muscle tension, cervical spine dysfunction, and vascular changes (Traumatic Brain Injury, 2023).ninds.nih
  • Sleep disorders affect thirty to seventy percent of traumatic brain injury patients and take various forms. Insomnia, characterized by difficulty initiating or maintaining sleep, affects approximately twenty-nine percent of patients. Sleep apnea, where breathing repeatedly stops during sleep, occurs in about twenty-five percent. Hypersomnia, excessive sleepiness or prolonged sleep need, affects twenty-eight percent. Narcolepsy develops in approximately four percent. These sleep disturbances result from damage to brain structures regulating sleep-wake cycles, hormonal disruptions affecting sleep, pain interfering with rest, and mood disturbances. Poor sleep quality worsens cognitive function, mood, pain, fatigue, and overall recovery (Impact of TBI on sleep, 2019).pmc.ncbi.nlm.nih
  • Depression emerges as a frequent complication, affecting thirteen to fifty-three percent of traumatic brain injury survivors. Post-traumatic depression may result from direct brain damage affecting mood-regulating circuits, particularly in frontotemporal regions, or from psychological response to injury-related losses and life changes. Symptoms include persistent sadness, loss of interest, feelings of worthlessness, guilt, changes in appetite and sleep, fatigue, difficulty concentrating, psychomotor agitation or retardation, and suicidal ideation. Depression significantly impairs rehabilitation participation, functional recovery, and quality of life. It increases caregiver burden and raises risk of suicide (Mood Disorders Following TBI, 2025).pmc.ncbi.nlm.nih+1
  • Anxiety disorders develop in eleven to seventy percent of traumatic brain injury patients. Post-traumatic stress disorder occurs particularly in those whose injuries resulted from violence, combat, or accidents. Generalized anxiety disorder involves excessive, uncontrollable worry about multiple life domains. Panic disorder includes unexpected panic attacks with physical symptoms like rapid heartbeat, sweating, trembling, and fear of dying. Social anxiety involves fear of social situations and negative evaluation. Anxiety often co-occurs with depression and exacerbates cognitive symptoms, sleep problems, and pain (Anxiety and Depression Following TBI, 2023).connectivity+1
  • Irritability and emotional dysregulation commonly follow traumatic brain injury, resulting from damage to frontal lobe regions that regulate emotions and control impulses. Individuals may experience frequent anger, decreased frustration tolerance, emotional outbursts disproportionate to triggers, rapid mood shifts, and difficulty calming down once upset. These symptoms strain relationships, interfere with community reintegration, and may limit employment options (Traumatic brain injury and mood disorders, 2020).pmc.ncbi.nlm.nih
  • Sensory sensitivities develop in many traumatic brain injury survivors. Light sensitivity, or photophobia, makes normal lighting uncomfortable and can trigger headaches. Noise sensitivity causes ordinary sounds to seem overwhelmingly loud or irritating. Some individuals develop increased sensitivity to touch, temperature, or smells. These sensitivities result from altered sensory processing in the injured brain and often accompany headaches and cognitive symptoms. They limit participation in bright or noisy environments and affect quality of life (Traumatic Brain Injury, 2023).ninds.nih

Beyond the Surface: Understanding the Effects of Personal Injury- Video

Integrative Approach: Chiropractic and Nurse Practitioner Care

An integrative approach combining chiropractic care with nurse practitioner oversight offers comprehensive support for individuals recovering from traumatic brain injuries. This collaborative model addresses the complex, multifaceted nature of brain injury by bringing together complementary expertise and treatment approaches. Chiropractic care focuses on the nervous system and musculoskeletal health through manual therapies, rehabilitative exercises, and supportive interventions. Chiropractors assess and address spinal alignment, particularly in the cervical spine which commonly sustains injury alongside traumatic brain injury. Cervical spine dysfunction contributes to many post-traumatic brain injury symptoms, including headaches, neck pain, dizziness, balance problems, and potentially cognitive symptoms through effects on blood flow and proprioceptive input (Chiropractic Care Supports TBI Healing, 2009).pinnaclehealthchiro

  • Spinal adjustments form a core component of chiropractic care for traumatic brain injury patients. These precise, controlled movements applied to spinal joints aim to restore proper alignment, reduce nerve interference, and optimize nervous system function. In the cervical spine, adjustments may improve blood flow to the brain by reducing compression on vertebral arteries. Research demonstrates that correction of cervical lordosis associates with immediate increases in cerebral blood flow, suggesting that biomechanical improvements can positively affect brain perfusion (Cervical lordosis correction increases cerebral blood flow, 2019).pmc.ncbi.nlm.nih
  • Chiropractic care addresses cerebrospinal fluid circulation, which plays important roles in brain health and recovery. Cerebrospinal fluid protects and nourishes the brain, removes metabolic waste products, and facilitates nutrient delivery. Traumatic brain injury can disrupt normal cerebrospinal fluid flow. Manual chiropractic techniques and spinal adjustments may help restore optimal cerebrospinal fluid dynamics, supporting brain healing processes (Chiropractic Care Supports TBI Healing, 2009).pinnaclehealthchiro
  • Soft tissue therapies complement spinal adjustments by addressing muscle tension, fascial restrictions, and myofascial pain that commonly develop after trauma. Techniques include manual therapy, instrument-assisted soft tissue mobilization, therapeutic massage, and trigger point release. These interventions reduce pain, improve tissue quality, restore range of motion, and promote healing. Addressing soft tissue dysfunction proves particularly important for cervical and upper thoracic regions where muscle tension contributes to headaches and affects cervical spine mechanics (Chiropractic Care Supports TBI Healing, 2009).elpasochiropractorblog+1

Rehabilitative exercises form an essential component of chiropractic traumatic brain injury care. Exercise programs address specific deficits identified through comprehensive assessment. Cervical strengthening and stabilization exercises improve neck muscle function and support spinal structures. Vestibular rehabilitation exercises help retrain balance systems through specific movements and visual tasks. Oculomotor exercises address eye tracking and coordination problems. Proprioceptive training improves body position awareness and motor control. Progressive strengthening builds overall fitness and supports functional recovery (Therapeutic Interventions for TBI, 2024).physio-pedia

Chiropractic neurologists employ specialized techniques to stimulate nervous system recovery. These approaches use specific sensory inputs, including light, sound, movement, and other stimuli, to activate targeted brain regions and promote neuroplasticity. The principle recognizes that the brain responds to appropriate stimulation similarly to how muscles respond to exercise, strengthening neural pathways through repeated activation. This non-invasive approach may benefit patients who have not responded well to traditional treatments (Chiropractic Neurology Supports Brain Healing, 2025).hmlfunctionalcare+1 Nurse practitioners provide medical oversight and management that complements chiropractic interventions. As board-certified family practice nurse practitioners with advanced training, they perform comprehensive health assessments, order and interpret diagnostic tests, prescribe medications when appropriate, manage medical comorbidities, and coordinate care across specialties. This medical oversight ensures that serious complications are identified and addressed promptly while supporting the body’s natural healing processes (Dr. Alex Jimenez’s Integrative Practice, n.d.).missionlegalcenter

  • Cognitive support represents an important aspect of nurse practitioner care for traumatic brain injury patients. Nurse practitioners assess cognitive function using standardized tools, provide education about cognitive symptoms and compensatory strategies, recommend cognitive rehabilitation services, and monitor cognitive recovery over time. They help patients and families understand cognitive changes and develop practical approaches to manage daily activities despite cognitive limitations (Strategies Nurses Use when Caring for Patients with TBI, 2019).nursing.duke+1
  • Metabolic and nutritional support provided by nurse practitioners addresses the altered metabolic demands following traumatic brain injury. The injured brain has increased energy requirements and specific nutritional needs. Nurse practitioners assess nutritional status, develop individualized nutrition plans, recommend supplements when indicated, and monitor response to nutritional interventions. Emerging research suggests that specific nutrients, including omega-3 fatty acids, vitamin D, magnesium, and amino acids, may support brain recovery when provided during the acute and subacute phases after injury (Nutritional interventions to support acute mTBI recovery, 2022).frontiersin
  • Emotional and psychological support forms another critical component of nurse practitioner care. They screen for depression, anxiety, post-traumatic stress disorder, and other mood disturbances, provide counseling and supportive therapy, prescribe psychotropic medications when appropriate, and refer to mental health specialists when needed. Addressing emotional health proves essential for overall recovery, as mood disturbances affect rehabilitation participation, cognitive function, and quality of life (Mood Disorders Following TBI, 2025).practicalneurology
  • Sleep management represents an area where nurse practitioner expertise particularly benefits traumatic brain injury patients. Nurse practitioners assess sleep quality and quantity, identify specific sleep disorders, recommend sleep hygiene improvements, prescribe sleep aids when appropriate, and coordinate sleep studies when indicated. Improving sleep quality enhances cognitive function, mood, pain management, and overall recovery (Sleep Disorders After Brain Injury, 2025).practicalneurology

The integrative model exemplified by Dr. Alexander Jimenez’s dual-scope practice demonstrates how chiropractic and nurse practitioner expertise can be combined within a single provider or collaborative team. Dr. Jimenez’s approach incorporates functional medicine principles, detailed health assessments, spinal and musculoskeletal care, nutritional support, exercise therapy, and comprehensive medical case management. This holistic model addresses the person as a whole rather than treating isolated symptoms, potentially leading to more complete and sustained recovery (Dr. Alex Jimenez’s Integrative Practice, n.d.).elpasochiropractorblog+1

Holistic Treatment Plans

Comprehensive, holistic treatment plans for traumatic brain injury incorporate multiple therapeutic approaches that address physical, cognitive, emotional, and metabolic aspects of recovery. These plans recognize that healing requires supporting the body’s natural recovery mechanisms while addressing specific symptoms and functional limitations.

  • Initial assessment forms the foundation of effective treatment planning. Comprehensive evaluation includes detailed history of the injury and symptom development, neurological examination assessing mental status, cranial nerves, motor function, sensory function, reflexes, coordination, and gait. Cervical spine assessment evaluates posture, range of motion, segmental mobility, muscle tone and strength, and joint function. Vestibular and oculomotor testing examines balance, eye movements, and visual-vestibular integration. Cognitive screening identifies attention, memory, processing speed, and executive function deficits. Mood and sleep questionnaires quantify emotional and sleep-related symptoms (Hidden TBI Symptoms: Integrative Model, 2025).elpasochiropractorblog
  • Spinal care targets cervical and upper thoracic dysfunction that commonly accompanies traumatic brain injury. Treatment begins with gentle mobilization techniques before progressing to specific adjustments as tolerated. The approach remains cautious, individualized, and responsive to patient symptoms, as excessive or aggressive treatment could worsen symptoms in vulnerable patients. Spinal adjustments aim to restore proper alignment, reduce nerve interference, improve proprioceptive input, and enhance blood flow to the brain. Treatment frequency and intensity adapt based on patient response, with some individuals benefiting from frequent initial visits that taper as function improves (Hidden TBI Symptoms: Integrative Model, 2025).zakerchiropractic+1
  • Soft tissue interventions address muscle tension, trigger points, fascial restrictions, and movement dysfunction. Manual therapy techniques include ischemic compression for trigger points, myofascial release for fascial restrictions, instrument-assisted soft tissue mobilization to address tissue quality, and therapeutic massage for overall relaxation and pain relief. These interventions reduce pain, improve tissue flexibility, restore normal movement patterns, and support overall healing. Treatment focuses particularly on cervical, upper thoracic, and cranial regions where soft tissue dysfunction contributes to headaches, neck pain, and dizziness (Hidden TBI Symptoms: Integrative Model, 2025).elpasochiropractorblog
  • Exercise therapy progresses systematically based on symptom tolerance and functional goals. Early-phase exercises focus on gentle range of motion, postural awareness, and basic strengthening within symptom limits. As tolerance improves, exercises advance to include cervical stabilization training to support injured structures, vestibular rehabilitation exercises to retrain balance systems, oculomotor exercises to improve eye coordination and tracking, proprioceptive training to enhance body position awareness, and progressive aerobic and strengthening exercises to build overall fitness. Exercise prescription follows graduated principles, starting with brief, low-intensity activities and progressing gradually while monitoring for symptom exacerbation (Hidden TBI Symptoms: Integrative Model, 2025).elpasochiropractorblog
  • Nutritional support addresses the increased metabolic demands and specific nutrient needs following traumatic brain injury. The injured brain requires adequate calories, high-quality protein for tissue repair, essential fatty acids particularly omega-3s for neural membrane health and anti-inflammatory effects, antioxidants to combat oxidative stress, vitamins and minerals for metabolic processes and neurotransmitter synthesis, and adequate hydration for optimal brain function. Nutritional assessment identifies deficiencies and guides supplement recommendations. Evidence suggests that omega-3 fatty acids, vitamin D, magnesium, and certain amino acids may support brain recovery when provided during acute and subacute phases (Nutritional interventions to support acute mTBI recovery, 2022).pmc.ncbi.nlm.nih+1
  • Cognitive rehabilitation strategies help individuals compensate for cognitive impairments and retrain affected abilities. Techniques include external memory aids such as written schedules, calendars, lists, and electronic reminders; attention strategies like reducing distractions, taking breaks, and focusing on one task at a time; organization systems that simplify and structure tasks; time management tools that help with planning and prioritization; and specific cognitive exercises that challenge and strengthen affected abilities. Education helps patients and families understand cognitive changes and develop realistic expectations while maintaining hope for continued improvement (Strategies Nurses Use when Caring for Patients with TBI, 2019).nursing.duke+1
  • Sleep optimization represents a crucial treatment component that affects multiple outcome domains. Sleep hygiene education covers maintaining consistent sleep-wake schedules, creating optimal sleep environments that are dark, quiet, and cool, limiting screen time before bed, avoiding caffeine and alcohol, and using relaxation techniques. Treatment of specific sleep disorders may include continuous positive airway pressure for sleep apnea, repositioning techniques for positional sleep disorders, or medications when appropriate. Improving sleep quality enhances cognitive function, mood, pain management, and overall recovery (Sleep Disorders After Brain Injury, 2025).practicalneurology
  • Pain management employs multiple modalities to address headaches, neck pain, and other pain complaints. Non-pharmacological approaches include manual therapy, therapeutic exercise, heat or cold application, relaxation techniques, and biofeedback. Pharmacological options when needed include appropriate analgesics, muscle relaxants, or nerve pain medications, used cautiously to avoid medication overuse and unwanted side effects. The goal emphasizes restoring function and reducing pain interference rather than complete pain elimination, which may not be achievable (Pain and Traumatic Brain Injury, 2024).health
  • Mood and emotional support acknowledges the profound psychological impacts of brain injury and chronic symptoms. Interventions include supportive counseling addressing adjustment to injury and life changes, cognitive-behavioral therapy to modify unhelpful thought patterns and behaviors, stress management techniques, mindfulness and relaxation training, support groups connecting individuals with others facing similar challenges, and psychiatric medications when appropriate. Family involvement and education play a crucial role in creating a supportive home environment that fosters recovery from mood disorders following traumatic brain injury (TBI) (Mood Disorders Following TBI, 2025).practicalneurology
  • Coordination across providers ensures comprehensive, efficient care. Regular communication between chiropractors, nurse practitioners, physical therapists, occupational therapists, speech-language pathologists, neuropsychologists, and other specialists involved in care promotes integrated treatment planning. Case conferences discuss patient progress, treatment responses, and plan modifications. This team approach prevents fragmented care and ensures all providers work toward shared goals. Dr. Alexander Jimenez’s practice exemplifies this coordination by offering multiple services within an integrated setting while maintaining referral relationships with specialists for needs beyond the clinic’s scope (Dr. Alex Jimenez’s Integrative Practice, n.d.).missionlegalcenter

Progress monitoring uses both objective measures and subjective reports to assess treatment effectiveness. Standardized outcome measures track symptom severity, functional abilities, and quality of life over time. Regular reassessments identify improvements, plateaus, or declines that necessitate adjustments to treatment. Flexible treatment plans adapt to changing needs as recovery progresses, with some interventions becoming less necessary as function improves, while others may need to be added or intensified. This responsive approach ensures that treatment remains appropriate throughout the recovery trajectory.

Conclusion

Through disturbed neuronal regulation, inflammatory processes, hormonal imbalances, and metabolic alterations, traumatic brain injury is a complicated medical disorder that affects not just the brain but the whole body. One of the most significant effects is cognitive impairment, which affects executive function, processing speed, memory, attention, and other mental skills essential for independent living. Autonomic dysfunction and systemic inflammatory reactions affect essential organs, whereas spasticity, weakness, coordination issues, and balance deficiencies affect the musculoskeletal system. Comprehensive treatment for traumatic brain injury requires an understanding of the brain-body link. Injury disrupts the complex communication networks that connect the brain to muscles, bones, organs, and metabolic systems, resulting in far-reaching impacts that necessitate a variety of therapeutic modalities. Healthcare professionals can identify the complete range of injury-related issues, including cognitive impairments that patients may not voluntarily mention and musculoskeletal or systemic symptoms that have a substantial impact on recovery, by obtaining a thorough history and asking methodical questions.

The multifaceted character of traumatic brain injury rehabilitation is addressed by an integrated strategy that combines chiropractic treatment with nurse practitioner supervision. Through soft tissue treatments, spinal adjustments, and rehabilitative activities that enhance biomechanics, lessen pain, and promote neuroplasticity, chiropractic interventions restore nervous system function. Nurse practitioners provide comprehensive care, including mood management, dietary counseling, medical supervision, cognitive support, and complete care coordination. Dr. Alexander Jimenez’s dual-scope practice exemplifies how this teamwork yields comprehensive treatment regimens that address the mental, emotional, physical, and metabolic aspects of recovery. Holistic treatment regimens include multiple therapy modalities that are customized to each patient’s specific needs and modified in response to those needs. While targeting specific symptoms and functional constraints, these programs recognize that promoting the body’s natural healing processes is crucial to a successful recovery. To promote healing and restore function, various therapies work in concert, including spinal care, soft tissue work, exercise therapy, nutritional assistance, cognitive rehabilitation, sleep optimization, pain management, and emotional support. Throughout the rehabilitation process, interventions are tailored to individual progress via progress tracking and therapeutic adaptability. Depending on a variety of circumstances, including age, location, severity of the damage, and pre-accident health, each person’s road to recovery from traumatic brain injury is unique. Some individuals heal very quickly and completely, while others have long-term functional impairments and lingering symptoms. By treating the whole person rather than just specific symptoms and by promoting the brain’s extraordinary ability to adapt and recover, an integrated, holistic approach gives promise for better results. Recovery prospects for those dealing with traumatic brain injury are expected to be substantially enhanced by ongoing research, better diagnostic instruments, and improved treatment modalities.

References

ChiroMed: Traumatic Brain Injury & Posture

ChiroMed: Traumatic Brain Injury & Posture

Traumatic Brain Injury & Posture: From Subtle Balance Changes to Abnormal Posturing — and How Integrative Chiropractic Care Can Help

Traumatic brain injuries (TBIs) can quietly change how you balance and stand, even months after a mild concussion. In the most serious cases, TBIs can trigger rigid reflex body positions called decorticate or decerebrate posturing, which are medical emergencies. These posture changes often stem from problems in how the brain uses sensory, visual, and vestibular (inner ear) signals. Neck and upper-back (cervical and upper thoracic) strain can exacerbate the problem by disrupting head-neck alignment and irritating nerves, which may worsen headaches and dizziness. An integrative plan that includes medical oversight, chiropractic adjustments, and sensory–motor therapies may help restore better alignment, reduce symptom drivers, and support safer balance over time (as part of a team approach). Mount Sinai Health System+3braininjurycanada.ca+3Brain Injury Association of America+3


Why TBIs Affect Posture

The brain’s balance triangle: vision, vestibular system, and body sense

Good balance depends on three main inputs working together: eyes (vision), the inner ear (vestibular system), and proprioception (your body’s internal sense of position). After a TBI, even a mild one, the brain may process these signals less efficiently. That can leave you feeling unsteady, dizzy, or “off,” especially during walking, turning the head, or in busy visual settings (like grocery aisles). Large groups of people with brain injuries report issues with balance, showing how common this problem can be. (Brain Injury Canada explains that balance integrates strength, vision, and inner-ear function and that balance problems are frequently reported after brain injury.) braininjurycanada.ca

Mild TBI: subtle but persistent postural-control changes

Research reviews show that after a concussion, people can have lingering deficits in postural control that routine tests sometimes miss. Nonlinear balance metrics and instrumented measures can detect differences even when symptoms appear to be improved. In other words, you might feel “almost fine,” but objective measures still pick up changes in sway, gait, or dynamic stability. PMC+1

Moderate to severe TBI: larger balance impairments

In moderate-to-severe TBI, studies document more obvious balance asymmetries and mobility limitations, which often require targeted, progressive rehab to improve safety and independence. OUP Academic


When Posture Becomes an Emergency: Abnormal Posturing

In rare but severe brain injuries, the body can assume reflex, rigid positions that signal deep brain dysfunction and require immediate medical care.

  • Decorticate posturing: arms flexed toward the chest with clenched fists; legs extended and rigid. It’s a sign of serious brain damage affecting pathways in the cerebral cortex, thalamus, or upper midbrain. Call emergency services at once if you see this. (Cleveland Clinic; Mount Sinai.) Cleveland Clinic+1
  • Decerebrate posturing: arms and legs extended, toes pointed down, head/neck arched backward, with rigid muscles—often linked to lower midbrain or pontine involvement. This also demands urgent care. (Cleveland Clinic; Mount Sinai.) Cleveland Clinic+1

Abnormal posturing is typically evaluated in conjunction with other signs using tools such as the Glasgow Coma Scale (GCS) during emergency assessments. NCBI


The Neck–Brain Link: How Cervical and Upper Thoracic Issues Can Worsen Symptoms

TBIs often occur with whiplash or neck strain, which can disturb joint motion, muscle tone, and head-on-neck position. In some patients, this can contribute to cervicogenic dizziness, headaches, and neck-related balance problems—especially when turning the head or maintaining upright posture. Clinical discussions from Dr. Jimenez’s team describe how cervical dysfunction and upper thoracic stiffness may aggravate dizziness and balance challenges after head/neck trauma. El Paso, TX Doctor Of Chiropractic+2El Paso, TX Doctor Of Chiropractic+2

  • Dr. Jimenez, DC, APRN, FNP-BC, emphasizes that a careful examination of posture, cervical range of motion, and joint motion can reveal overlooked factors contributing to headaches and dizziness, and that progress often includes cervical stabilization and vestibular drills, alongside other care. El Paso, TX Doctor Of Chiropractic+1

What Symptoms Might You Notice?

  • Feeling wobbly, light-headed, or “tilted,” especially in visually busy places
  • Headaches (often starting at the neck or base of the skull), neck pain, and eye strain
  • Dizziness when turning the head, rolling in bed, or after long screen time
  • Fatigue, brain fog, or irritability that worsens as the day goes on
  • Slower walking, shorter steps, or veering off line

These align with common post-concussion complaints (headache, dizziness, fatigue) and with mobility/balance challenges described in the brain-injury literature. PMC+1

Symptom Questionnaire:


How Integrative Chiropractic Care Can Fit Into a TBI Recovery Plan

Important: Chiropractic care does not treat the brain injury itself and should not replace medical diagnosis or urgent care. It may, however, support symptom management and functional recovery when coordinated with your medical team (neurology, primary care, vestibular/physical therapy). Bergeron Clifford LLP

1) Restoring better spinal mechanics and alignment (especially upper neck)

Gentle, carefully selected spinal adjustments can reduce joint restrictions and muscle guarding in the cervical and upper thoracic regions. For some patients, improving head–neck alignment can reduce neck-related headaches and dizziness, which can indirectly improve balance and posture. Dr. Jimenez’s clinical materials and other chiropractic sources describe these goals and report symptom relief in select cases where the neck is a contributing factor. El Paso, TX Doctor Of Chiropractic+2El Paso, TX Doctor Of Chiropractic+2

2) Supporting neurophysiology and fluid dynamics (theoretical/adjunctive)

Some clinics note that adjustments may improve blood and cerebrospinal fluid (CSF) circulation, potentially aiding brain recovery by optimizing the environment around neural tissue. The evidence here is preliminary and should be framed as “may help” within a broader rehabilitation plan; still, it’s a common adjunctive rationale in clinical practice. Impact Medical Group+1

3) Sensory–motor rehabilitation to rebuild coordination

Integrative chiropractic and functional-neurology clinics often pair adjustments with targeted sensory and movement therapies: gaze stabilization, saccade/pursuit drills, balance progressions (wide base → narrow base → head turns), dual-task walking, and cervical proprioception exercises. These aim to retrain the brain (neuroplasticity) and calibrate vision–vestibular–proprioceptive inputs. HML Functional Care

4) Team-based care improves outcomes and safety

Medical guidance identifies red flags, rules out dangerous causes, and directs imaging or vestibular testing when needed. Rehabilitation professionals measure postural control, gait, and mobility using validated tools to demonstrate progress over time. Observational and review data indicate that balance changes occur after concussion, supporting the need for a structured assessment to guide rehabilitation. PMC+1


A Step-By-Step Care Pathway (What This Can Look Like)

  1. Medical evaluation first (especially if symptoms are new, severe, or worsening). Providers check for red flags and determine whether urgent care or imaging is necessary. Abnormal posturing = emergency. Mount Sinai Health System+1
  2. Baseline function check: vision, vestibular function, neck exam, simple balance tests. archives-pmr.org
  3. Cervical and upper thoracic care: gentle mobilization/adjustments (as appropriate), soft-tissue work, and home exercises to restore motion and reduce headache/neck-related dizziness. El Paso, TX Doctor Of Chiropractic
  4. Sensory–motor retraining: vestibular and oculomotor drills, graded balance tasks, gait training; progress in small, safe steps. HML Functional Care
  5. Lifestyle and pacing: sleep, graded activity, hydration, and symptom-paced screens/exercise—often supported by nurse-practitioner-led coaching in integrative settings. (Dr. Jimenez’s practice materials emphasize whole-person plans and steady progression.) El Paso, TX Doctor Of Chiropractic

How TBIs Can Lead to Spinal Misalignments and Symptom Flares

  • Impact mechanics (falls, crashes, sports) can strain facet joints, discs, and deep neck muscles.
  • The body may then adopt protective postures (chin jutting, shoulder guarding), which can irritate cervical nerves and muscle trigger points.
  • These patterns may worsen headaches and dizziness by disturbing cervical proprioception and upper-neck mobility—especially around C0–C2, a frequent source of cervicogenic symptoms after whiplash/TBI. Clinical articles on cervicogenic dizziness echo these links and suggest appropriate manual care and stabilization when indicated (after medical clearance). El Paso, TX Doctor Of Chiropractic+1

When Symptoms Become “Rigid Posturing”

Remember: decorticate or decerebrate posturing means severe brain dysfunction. The person is typically unconscious and in a coma; both patterns require 911/emergency care now. (Do not attempt chiropractic or rehab; call for medical help immediately.) Cleveland Clinic+1


Tests and Tools for TBI & Postural Problems (From Simplest to Most Advanced)

Note: Your exact pathway depends on symptoms and safety. Start with medical evaluation and add tests as needed.

Bedside & Screening (simplest)

  • History and neuro exam (headache, dizziness, nausea, vision changes, sleep, mood, neck pain; cranial nerves; coordination).
  • Glasgow Coma Scale (GCS) in acute settings to rate eye, verbal, and motor responses. NCBI
  • Symptom scales (e.g., post-concussion symptom checklists). Mayo Clinic
  • Basic balance screens (Romberg, tandem stance, timed up-and-go), and observation of gait and turns.
  • Cervical exam: range of motion, segmental motion, palpation, and joint position error tests for proprioception when appropriate. (Dr. Jimenez highlights posture and cervical mechanics in clinical content.) El Paso, TX Doctor Of Chiropractic

Clinic-level functional tests

  • BESS (Balance Error Scoring System) and instrumented postural sway for more sensitive detection of balance deficits after concussion. PMC
  • Community Balance & Mobility Scale (CB&M) for higher-level balance and mobility challenges (validated in brain injury populations). PMC
  • Vestibular/Oculomotor screening (e.g., smooth pursuit, saccades, vestibulo-ocular reflex/gaze stabilization, visual motion sensitivity).
  • Cervical/vestibular differentiation tests (to help sort inner-ear vs. neck-driven dizziness).

Specialized vestibular & ocular testing

  • Videonystagmography (VNG), calorics, rotary chair, and dynamic visual acuity tests to quantify vestibular deficits.
  • Eye-tracking or computerized oculomotor measures for pursuit/saccades.
  • Computerized posturography/force-plate is utilized for objective sway and strategy analysis, while center-of-mass measures aid in characterizing dynamic postural control following a concussion. IJSPT

Neurocognitive assessment

  • Standardized tests of attention, processing speed, memory, and executive function are used in concussion management (clinic-dependent).

Imaging & electrophysiology (advanced)

  • CT (acute bleed/fracture) and MRI (structural injury).
  • Diffusion Tensor Imaging (DTI) (white-matter pathways) and functional MRI in research/selected clinical contexts.
  • EEG if seizures or atypical episodes are suspected. (Mount Sinai lists EEG among tests for abnormal posturing workups; emergency pathways decide timing.) Mount Sinai Health System+1
  • PET/SPECT in select specialty centers; blood biomarkers (e.g., GFAP, UCH-L1) may be used in emergency algorithms.

Evidence Snapshots: What Research and Clinical Sources Say

  • Postural control can remain impaired after concussion; sophisticated metrics can reveal deficits not obvious on quick screens. PMC
  • Dynamic postural control, as measured by center-of-mass, is a useful outcome within one year post-concussion. IJSPT
  • Balance limitations after TBI are common and affect independence; better sitting balance early in rehab predicts better self-care after discharge. Brain Injury Association of America
  • Cervicogenic dizziness and neck-related headache can follow whiplash/head trauma; carefully managed manual therapy and cervical stabilization may reduce symptom drivers. (Clinical sources, including Dr. Jimenez’s site.) El Paso, TX Doctor Of Chiropractic+1
  • Chiropractic care should be adjunctive—not a replacement for medical treatment—and may help selected patients as part of a team plan, especially when cervical dysfunction contributes to symptoms. Bergeron Clifford LLP
  • Some clinics suggest that adjustments may help with blood and cerebrospinal fluid flow; however, this idea remains a theory and should be clearly explained to patients and used as part of a medically supervised plan. Impact Medical Group+1

A Practical, Integrated Plan (Example)

Built around safety, simplicity, and steady progress—and coordinated with your medical team.

  1. Protect & screen: See a clinician first. Urgent signs (worsening severe headache, repeated vomiting, loss of consciousness, new weakness/vision loss, abnormal posturing) need emergency care. Mount Sinai Health System+1
  2. Calm the neck: Gentle manual therapy and mobility work for the cervical/upper thoracic regions to reduce joint restriction and muscle guarding. Add home drills (chin nods, scapular setting, breathing) and progress slowly. El Paso, TX Doctor Of Chiropractic
  3. Recalibrate balance systems: Start with a wide-base stance, eyes open → eyes closed; then narrow base; then add head turns and dual-task steps. Integrate gaze stabilization (VOR) and visual motion tolerance exercises as symptoms allow. HML Functional Care
  4. Train real-life tasks: Gentle walking on level ground → turns → uneven terrain; keep sessions short and frequent. Measure progress with CB&M or instrumented sway when available. PMC
  5. Whole-person support: Sleep regularity, hydration, anti-inflammatory nutrition, and pacing (breaks between screens/reading). Clinics like Dr. Jimenez’s emphasize collaborative care—chiropractic care, nurse practitioner oversight, and vestibular/physical therapy—ensuring each domain is covered. El Paso, TX Doctor Of Chiropractic

When to Call Right Away (Red Flags)

  • Abnormal posturing (decorticate/decerebrate), severe confusion, or unresponsiveness
  • Worsening severe headache, repeated vomiting, seizures, new weakness/numbness, or vision loss
  • Neck pain with fever, sudden stiff neck, or neurological deficits

These signs need emergency evaluation—not clinic-based care. Mount Sinai Health System+1


How Dr. Alexander Jimenez’s Team Applies This Locally (El Paso)

Dr. Jimenez, DC, APRN, FNP-BC, highlights a dual-scope approach: identifying cervical drivers of headache/dizziness, rebuilding posture with gentle adjustments and stabilization, and combining this with vestibular drills, balance progressions, and lifestyle support. His clinical articles emphasize the importance of careful posture and cervical motion exams, stepwise progress, and collaborative plans with medical and rehabilitation partners. El Paso, TX Doctor Of Chiropractic+1


The Bottom Line

  • Mild TBI can leave behind subtle balance problems; severe TBI can cause abnormal posturing—an emergency. PMC+2Cleveland Clinic+2
  • These changes stem from how the brain integrates vision, vestibular input, and body sense, and they can be worsened by neck/upper-back dysfunction. braininjurycanada.ca+1
  • Integrative care—encompassing medical oversight, targeted chiropractic adjustments for cervical mechanics, and sensory–motor rehabilitation—offers a practical path to safer posture and stability. HML Functional Care+1

References

Brain Injury Association of America. (n.d.). [Factors associated with sitting and standing balance]. https://biausa.org/ Brain Injury Association of America

Brain Injury Association of America. (n.d.). [Sitting balance in rehabilitation is a good predictor of the amount of assistance that will be required]. https://biausa.org/ Brain Injury Association of America

Brain Injury Canada. (n.d.). [Balance]. https://braininjurycanada.ca/ braininjurycanada.ca

Brain Injury Canada. (n.d.). [Mobility]. https://braininjurycanada.ca/ braininjurycanada.ca

Cleveland Clinic. (2023, May 9). [Decerebrate posturing: What it is, causes, & treatment]. https://my.clevelandclinic.org/ Cleveland Clinic

Cleveland Clinic. (2023, May 9). [Decorticate posturing: What it is, causes, & treatment]. https://my.clevelandclinic.org/ Cleveland Clinic

Inness, E. L., et al. (2011). [Measuring balance and mobility after traumatic brain injury: Validation of the Community Balance and Mobility Scale (CB&M)]. Journal of Neurosurgery, 114(6). https://pmc.ncbi.nlm.nih.gov/ PMC

Mount Sinai Health Library. (2025, Apr 16). [Decerebrate posture]. https://www.mountsinai.org/ Mount Sinai Health System

Mount Sinai Health Library. (2025, Apr 16). [Decorticate posture]. https://www.mountsinai.org/ Mount Sinai Health System

Patejak, S., et al. (2021). [A systematic review of center of mass as a measure of dynamic postural control following concussion]. International Journal of Sports Physical Therapy. https://ijspt.scholasticahq.com/ IJSPT

Permenter, C. M., et al. (2023). [Postconcussive syndrome]. StatPearls. https://www.ncbi.nlm.nih.gov/books/ NCBI

Sosnoff, J. J., et al. (2011). [Previous mild traumatic brain injury and postural-control dynamics]. Journal of Athletic Training. https://pmc.ncbi.nlm.nih.gov/ PMC

Buckley, T. A., et al. (2016). [Postural control deficits identify lingering post-concussion neurological deficits]. Journal of Athletic Training. https://pmc.ncbi.nlm.nih.gov/ PMC

Jain, S., et al. (2023). [Glasgow Coma Scale]. StatPearls. https://www.ncbi.nlm.nih.gov/books/ NCBI

Flint Rehab. (2021). [Posturing after brain injury: Types and recovery outlook]. https://www.flintrehab.com/ Flint Rehab

HML Functional Care. (2025, Jul 22). [How chiropractic neurology supports brain healing]. https://hmlfunctionalcare.com/ HML Functional Care

Impact Medical Group. (2024, Jun 26). [Can chiropractic care help with mild traumatic brain injuries?] https://www.impactmedicalgroup.com/ Impact Medical Group

Northwest Florida Physicians Group. (2025). [Using chiropractic care to treat traumatic brain injuries]. https://northwestfloridaphysiciansgroup.com/ Northwest Florida Physicians Group

Pinnacle Health Chiropractic. (2025). [Six ways chiropractic care supports healing after TBI]. https://www.pinnaclehealthchiro.com/ pinnaclehealthchiro.com

ThinkVida. (2025). [Treating concussions with chiropractic care]. https://thinkvida.com/ Vida Integrated Health

Jimenez, A. (n.d.). [Finding hidden TBI symptoms: Signs you might miss]. dralexjimenez.com. https://dralexjimenez.com/ El Paso, TX Doctor Of Chiropractic

Jimenez, A. (n.d.). [Neck pain and feeling dizzy: Cervicogenic/cervical vertigo]. dralexjimenez.com. https://dralexjimenez.com/ El Paso, TX Doctor Of Chiropractic

Jimenez, A. (n.d.). [Cervicogenic dizziness from whiplash]. dralexjimenez.com. https://dralexjimenez.com/ El Paso, TX Doctor Of Chiropractic

Jimenez, A. (2025). [Traumatic brain injury: Understanding the long-term effects]. dralexjimenez.com. https://dralexjimenez.com/ El Paso, TX Doctor Of Chiropractic


Hidden Traumatic Brain Injury Symptoms: Signs Missed

Hidden Traumatic Brain Injury Symptoms: Signs Missed

Hidden Traumatic Brain Injury (TBI) Symptoms: How an Integrative Chiropractic + Nurse Practitioner Team Finds What Others Miss

Overview

Many traumatic brain injuries (TBIs)—especially mild TBIs or concussions—go unnoticed at first. Symptoms can be subtle, delayed, or brushed off as stress, fatigue, or “just getting older.” A careful clinician can catch what others miss by taking a thorough patient history and asking targeted questions that explore cognitive, emotional, sensory, sleep, and balance changes. (Mayo Clinic, n.d.; BrainLine, 2017). Mayo Clinic+1

This article explains:

  • A chiropractor or nurse practitioner may uncover hidden symptoms through a thorough history and structured questioning.
  • Why TBIs get missed, and how to avoid that.
  • A step-by-step diagnostic ladder, from basic screens to advanced tools, matched to symptom complexity.
  • An integrative care plan, combining chiropractic care for the spine, neck, and vestibular system with nurse practitioner (NP) medical oversight for whole-person recovery.

We also provide clinical insights that align with the combined approach of Dr. Alexander Jimenez, DC, APRN, FNP-BC, who focuses on thorough patient history, functional exams, and gradual plans for returning to work and activities (DrAlexJimenez.com; LinkedIn). El Paso, TX Doctor Of Chiropractic+1


Why TBIs Are Easy to Miss

  1. Symptoms can be delayed or vague. People may notice headaches, brain fog, irritability, or sleep changes days or weeks after the event. Sensory issues such as changes in smell or taste and sensitivity to light or noise also occur, and patients often don’t connect them to a past bump, crash, or whiplash. (BrainLine, 2017; Mayo Clinic, n.d.). BrainLine+1
  2. Imaging can be normal. Standard CT or MRI may look fine in mild TBI, yet symptoms persist. That’s why history and examination are crucial—and why advanced tools are sometimes needed later. (Mayo Clinic, n.d.). Mayo Clinic
  3. Invisible wounds. Military and civilian clinicians stress that TBIs often present as “invisible injuries.” Without active screening, they are easy to overlook. (Hanscom AFB/AFMS; Health.mil). Hanscom Air Force Base+1

Hidden Symptoms To Ask About (And Why)

A skilled chiropractor or NP will conduct a thorough examination. Along with open-ended conversation, they use symptom checklists and guided probes that reveal patterns across body systems.

Cognitive and emotional

  • Trouble focusing, slowed thinking, memory lapses, “losing the thread” mid-task
  • Irritability, mood swings, anxiety, or depression
  • Feeling “not like myself,” “foggy,” or overwhelmed in busy environments
    (BrainLine, 2017). BrainLine

Sensory

  • Loss or change in smell or taste
  • Light/noise sensitivity; blurred vision; “seeing stars”
  • Ringing in the ears (tinnitus)
    (BrainLine, 2017). BrainLine

Physical

  • Headaches (especially new, worsening, or “pressure-type”)
  • Dizziness, vertigo, balance problems, coordination changes
  • Fatigue; neck pain that worsens with screens or reading
    (Mayo Clinic, n.d.; BrainLine, 2017). Mayo Clinic+1

Sleep and autonomic

  • Difficulty falling or staying asleep; unusual daytime drowsiness
  • Symptoms include orthostatic intolerance, which causes lightheadedness upon standing, as well as palpitations and heat or cold intolerance.
    (Mayo Clinic, n.d.). Mayo Clinic

Key point: These symptoms are common after mild TBI—even with a normal CT—and they often overlap. A structured, curious interview is the quickest path to the right diagnosis. (Mayo Clinic, n.d.; Hanscom AFB). Mayo Clinic+1


The Power of a Thorough History: What to Ask

Example of Symptom Questionnaire:

Below is a practical set of targeted questions clinicians use to uncover hidden TBI patterns. Patients and families can use this as a self-checklist to bring to appointments.

Mechanism and timeline

  • What happened? (fall, car crash, sports, blast, whiplash, strike to head/neck?)
  • Did you black out, feel dazed, or lose memory of events?
  • When did symptoms begin—immediately, hours later, or days later? (Mayo Clinic, n.d.). Mayo Clinic

Headache and neck

  • New or changing headaches? What triggers them (screens, reading, exercise, lack of sleep)?
  • Neck pain or stiffness, pain during head movements, and neck fatigue throughout the day are all associated with cervicogenic headaches and vestibular problems. (Mayo Clinic, n.d.; BrainLine, 2017). Mayo Clinic+1

Cognition and mood

  • Are you experiencing difficulty concentrating, slowed processing, or short-term memory slips?
  • Are you experiencing irritability, mood swings, anxiety, depression, or emotional “numbness”? (BrainLine, 2017; Health.mil). BrainLine+1

Sensory

  • Has there been a change in your sense of smell or taste?
  • Have you noticed any new sensitivity to light or noise, experienced blurred or double vision, or experienced eye strain when reading? (BrainLine, 2017). BrainLine

Balance and dizziness

  • Dizziness, vertigo, poor balance, and motion sensitivity (in a car or in a store) are common symptoms. Falls? (BrainLine, 2017). BrainLine

Sleep

  • Trouble falling asleep, frequent waking, and feeling unrefreshed? (BrainLine, 2017; Mayo Clinic, n.d.). BrainLine+1

Function and safety

  • Are you comfortable driving at night or at high speeds on the highway?
  • Screen tolerance (work, school, phone)?
  • Return to work/sport issues?

Red flags (urgent referral)

  • Symptoms that require urgent referral include worsening headache, repeated vomiting, weakness or numbness, slurred speech, seizures, extreme drowsiness, new confusion, and unequal pupils. (Mayo Clinic, n.d.). Mayo Clinic

Where Chiropractic Care Fits (with NP Supervision)

Chiropractors often see patients after car crashes, sports injuries, and falls. They evaluate the cervical spine, posture, proprioception, and vestibular-ocular systems—all of which can drive headaches, dizziness, and cognitive fatigue after TBI. A growing body of interprofessional work suggests that chiropractors can play a role in screening, referral, and rehabilitative care for concussion-related neck and balance disorders, especially when working as part of a team. (NW Health/Chiropractic Economics piece; peer commentary on chiropractors’ role in SRC). Northwestern Health Sciences University+1

Nurse practitioners provide medical oversight, screen for red flags, coordinate imaging and lab tests, and manage sleep, mood, metabolic, and medication issues that often complicate recovery. Nursing literature emphasizes neuromonitoring, family education, and prevention of secondary injury, even outside the ICU. (Figueiredo et al., 2024). MDPI

A collaborative care model improves symptom tracking and coordination—especially for chronic pain and persistent symptoms after TBI. (Curran et al., 2024; Ilkhani et al., 2024). PMC+1

Clinical note (consistent with Dr. Jimenez’s approach): Combine careful history and targeted exams with staged spinal care, vestibular/oculomotor rehab, aerobic re-conditioning, and nutrition/sleep coaching—while the NP manages medical needs and coordinates imaging or biomarkers when indicated. (DrAlexJimenez.com). El Paso, TX Doctor Of Chiropractic


Diagnostic Tools for TBI: From Basic to Advanced

Think of assessment as a ladder. Start simple; climb only as needed, based on red flags, symptom persistence, and functional limits.

1) Basic bedside screening (every visit)

  • Symptom scales
    • PCSS (Post-Concussion Symptom Scale) – quick 22-item rating; easy to trend over time. (Intermountain Health PDF; Langevin et al., 2022). Intermountain Healthcare+1
    • RPQ (Rivermead Post-Concussion Symptoms Questionnaire) – useful if scored as RPQ-3 and RPQ-13 subscales. (Eyres et al., 2005; Zeldovich et al., 2023). PubMed+1
  • Sport Concussion Assessment Tool (SCAT5) – standardized sideline/clinic tool (13+ years); includes PCSS, balance, and cognitive screens. (BJSM SCAT5; BMX SCAT5). British Journal of Sports Medicine+1
  • Cognitive screen
    • MoCA (Montreal Cognitive Assessment) – sensitive for subtle deficits; faster and more sensitive than MMSE in TBI populations. (Waldron-Perrine et al., 2019). PMC
  • Vestibular-ocular screen
    • VOMS – brief test provoking symptoms with pursuits/saccades, near-point convergence, and vestibulo-ocular reflex. Highly practical after a concussion. (Mucha et al., 2014). PMC
  • Balance
    • BESS (Balance Error Scoring System) – simple stance tests scored by errors. (NCAA/Atrium manuals; APTA summary). fs.ncaa.org+2Atrium Health+2
  • Cranial nerve + smell/taste queries
    • Ask directly about smell/taste changes, and test if possible. These sensory shifts are common but under-reported. (BrainLine, 2017). BrainLine

Why this matters: Many mild TBIs won’t show on CT/MRI. These low-cost tools at the point of care catch patterns and guide next steps. (Mayo Clinic, n.d.). Mayo Clinic


2) Intermediate testing (when symptoms persist or are complex)

  • Comprehensive vestibular assessment
    • Videonystagmography (VNG), oculomotor testing, and computerized dynamic posturography / Sensory Organization Test (SOT) to quantify balance control and track rehab response. (UHC policy summary; RehabMeasures; related trial). UHC Provider+2Shirley Ryan AbilityLab+2
  • Neurocognitive testing
    • If cognitive loads (work, school, and driving) remain limited, consider using formal batteries (clinic-based or computerized). (SCAT5 framework). British Journal of Sports Medicine
  • Mental health screening
    • Depression, anxiety, and PTSD screens to address “invisible” sequelae early—important for prognosis and adherence. (Health.mil). Military Health System

3) Advanced diagnostics (selected cases)

  • Conventional neuroimaging
    • Non-contrast CT for acute red flags (rule out bleed/skull fracture).
    • MRI (with appropriate sequences) if symptoms persist or focal deficits appear. (Mayo Clinic, n.d.). Mayo Clinic
  • Advanced MRI sequences
    • DTI (Diffusion Tensor Imaging): detects white matter microstructural changes not seen on routine MRI; can improve prognostic models in mTBI with normal CT. (Patil et al., 2025; Richter et al., 2024; Paolini et al., 2025). PMC+2The Lancet+2
    • SWI (Susceptibility-Weighted Imaging): sensitive to traumatic microbleeds and diffuse axonal injury; the presence of microbleeds may relate to persistent complaints in some patients. (Hsu et al., 2023; Hageman et al., 2022; Eldeş et al., 2020). PubMed+2PubMed+2
    • fMRI (task-based or resting-state): research and selected clinical programs use it to map functional disruptions after concussion. (Irimia et al., 2015; Jantzen et al., 2004). PMC+1
  • Electrophysiology
    • EEG/qEEG plays an evolving role in detecting or monitoring changes in networks associated with traumatic brain injury (TBI) and should be conducted according to professional guidelines, with interpretations placed in a clinical context. (Haneef et al., 2013; ACNS guideline, 2020; Stevens et al., 2024). PMC+2acns.org+2
  • Blood biomarkers
    • Blood tests for GFAP and UCH-L1 are FDA-approved to help determine whether adults with suspected mild traumatic brain injury need a CT scan, and labs are now offering these tests (JAMA Netw Open, 2024; bioMérieux press release, 2024). JAMA Network+1

Bottom line: Start with history and bedside tools. Escalate to advanced testing when symptoms persist, red flags emerge, or functional demands require precise guidance.


The Integrative Plan: Chiropractic + Nurse Practitioner

Goals

  1. Reduce symptoms (headache, dizziness, neck pain, cognitive fatigue).
  2. Restore systems (cervical, vestibular-ocular, autonomic).
  3. Rebuild capacity (sleep, mood, fitness, cognition).
  4. Return to life (drive, work/school, sports) with safe progressions.

Chiropractic care (examples)

  • Cervical spine evaluation and treatment to reduce neck-driven headaches and improve proprioception—often key for balance and eye-head coordination. (NW Health/Chiropractic Economics; Denver Chiropractic overview). Northwestern Health Sciences University+1
  • Vestibular and oculomotor exercises (gaze stabilization, smooth pursuits, saccades, and convergence work) were built from VOMS findings. (Mucha et al., 2014). PMC
  • Soft-tissue therapy and graded mobility to decrease pain-guarding patterns and improve movement tolerance for daily tasks.

Note: Some clinics describe additional mechanisms (e.g., effects on CSF flow). Evidence for such claims varies, and treatment plans should focus on function, symptoms, and measurable gains. (Pinnacle; Apex; NorthWest Florida Physicians Group). pinnaclehealthchiro.com+2Apex Chiropractic+2

Nurse practitioner oversight

  • Medical screening & safety: identify red flags; determine need for CT/MRI; manage post-traumatic headache, sleep issues, and mood symptoms. (Mayo Clinic; Figueiredo et al., 2024). Mayo Clinic+1
  • Metabolic support: address blood pressure, glucose, thyroid, anemia, hydration, and nutrition that affect brain recovery; coordinate referrals. (Figueiredo et al., 2024). MDPI
  • Education and pacing should guide cognitive and physical pacing, facilitate a graded return to tasks, and provide family support. (Health.mil; Figueiredo et al., 2024). Military Health System+1

Collaborative care pays off. TBI programs using team-based models show better coordination and patient-centered outcomes, especially when pain and mood complicate recovery. (Curran et al., 2024; Ilkhani et al., 2024). PMC+1


How a Thorough Approach Uncovers the Missed Diagnosis

  1. History finds the pattern. A patient with “new headaches and irritability” might also report loss of smell, motion sensitivity in stores, and neck stiffness—indicating strong post-concussive and cervical/vestibular involvement. (BrainLine, 2017). BrainLine
  2. Bedside tests confirm direction. An abnormal VOMS (symptom spikes on saccades or VOR) and BESS errors cement the vestibular-ocular target for therapy. (Mucha et al., 2014; NCAA/Atrium). PMC+1
  3. Escalate only when needed. If symptoms persist despite progress—or if work/sport demands are high—consider advanced MRI (DTI/SWI), qEEG, or biomarkers to refine prognosis and guide next steps. (Patil et al., 2025; Hsu et al., 2023; ACNS, 2020; JAMA, 2024). JAMA Network+3PMC+3PubMed+3

A Practical, Staged Care Roadmap

This is a general template. Your plan should be individualized based on findings and safety.

Weeks 0–2: Calm and orient

  • Education on pacing, hydration, and sleep hygiene; light neck mobility; sub-symptom aerobic activity (e.g., easy walks).
  • Begin cervical care and gentle vestibular/oculomotor drills if tolerated.
  • NP manages headache/sleep, screens mood, and ensures no red flags. (Mayo Clinic; Figueiredo et al., 2024). Mayo Clinic+1

Weeks 2–6: Re-train systems

  • Progress cervical stabilization and posture work; expand gaze stabilization and convergence tasks; add balance progressions.
  • Short bouts of cognitive-physical dual tasking (e.g., reciting while walking) as symptoms allow.
  • Use PCSS or RPQ weekly to track trend lines. (Intermountain PCSS; Eyres et al., 2005). Intermountain Healthcare+1

Weeks 6–12: Build capacity

  • Increase aerobic exercise toward moderate intensity; integrate return-to-drive and return-to-work checklists.
  • If plateaus persist, consider intermediate/advanced assessments (SOT/posturography; DTI/SWI in selected cases). (RehabMeasures SOT; Patil et al., 2025). Shirley Ryan AbilityLab+1

Beyond 12 weeks: Persistent symptoms

  • Titrate therapies; address mood/sleep/autoimmune or endocrine drivers; consider collaborative pain programs. (Curran et al., 2024). PMC

Where Local Chiropractic or Functional Neurology Clinics Fit

Community clinics frequently educate patients about post-concussion care and offer combined chiropractic + vestibular/oculomotor programs under medical supervision. These clinics emphasize neck care, balance/eye-movement drills, and staged activity. (Denver Chiropractic; Calibration Chiropractic + Functional Health; HML Functional Care). Denver Integrated Spine Center+2calibrationmansfield.com+2

Clinical observation (aligned with Dr. Jimenez’s posts): Patients often report that a combined neck + vestibular/ocular approach reduces headache frequency, steadies vision, and improves stamina for work or driving. (DrAlexJimenez.com). El Paso, TX Doctor Of Chiropractic


Safety Reminders

  • If you develop a worsening headache, repeated vomiting, seizure, weakness, confusion, or unequal pupils, seek emergency care immediately. (Mayo Clinic, n.d.). Mayo Clinic
  • Spinal manipulation is not used in unstable injuries. Care should follow a full exam, with imaging or referrals when indicated.

Take-Home Messages

  • Hidden symptoms are common after TBI. They span thinking, mood, senses, sleep, and balance. (BrainLine, 2017; Mayo Clinic, n.d.). BrainLine+1
  • A thorough history and targeted questions are the most powerful diagnostic tools.
  • Use a ladder of tests, from PCSS/RPQ, VOMS, BESS, and MoCA to SOT, advanced MRI (DTI/SWI), EEG/qEEG, and GFAP/UCH-L1 biomarkers, based on complexity. (Mucha et al., 2014; ACNS, 2020; JAMA, 2024; Patil et al., 2025). PMC+3PMC+3acns.org+3
  • An integrative teamchiropractor + NP—covers structure, neurology, and overall health, improving safety and continuity of care. (Figueiredo et al., 2024; Curran et al., 2024). MDPI+1

References

BrainLine. (2017, December 1). Symptoms of traumatic brain injury (TBI). BrainLine

BrainLine. (2017, June 21). TBI 101: Physical symptoms. BrainLine

Curran, M. C., et al. (2024). Chronic pain after traumatic brain injury: A collaborative care intervention (TBI Care). PMC

Eyres, S., Carey, A., Gilworth, G., Neumann, V., & Tennant, A. (2005). Construct validity and reliability of the Rivermead Post-Concussion Symptoms Questionnaire (RPQ). PubMed

Figueiredo, R., Castro, C., & Fernandes, J. B. (2024). Nursing interventions to prevent secondary injury in critically ill patients with traumatic brain injury: A scoping review. Journal of Clinical Medicine, 13(8), 2396. MDPI

Haneef, Z., Levin, H., & Masel, B. (2013). Electroencephalography and quantitative EEG in mild traumatic brain injury. PMC

Hanscom Air Force Base Public Affairs. (2017, March 17). TBI recognition critical to treating invisible wounds. Hanscom Air Force Base

Health.mil. (2022, February 4). Air Force Invisible Wounds Initiative helps build a supportive culture. Military Health System

Hsu, C. C. T., et al. (2023). The current state of susceptibility-weighted imaging and its clinical applications in TBI. PubMed

Ilkhani, S., et al. (2024). Beyond surviving: A scoping review of collaborative care models to inform the future of post-discharge trauma care. PMC

Intermountain Health. (2024). Post-Concussion Symptom Scale (PCSS). Intermountain Healthcare

Irimia, A., et al. (2015). Functional neuroimaging of traumatic brain injury: Advances and clinical utility. PMC

JAMA Network Open. (2024). Papa, L., et al. Diagnostic performance of GFAP, UCH-L1, and MAP-2 for TBI evaluation. JAMA Network

Jantzen, K. J., et al. (2004). A prospective fMRI study of mild traumatic brain injury. PMC

Mayo Clinic. (n.d.). Traumatic brain injury—Symptoms & causes. Mayo Clinic

Mayo Clinic. (n.d.). Traumatic brain injury—Diagnosis & treatment. Mayo Clinic

Mucha, A., et al. (2014). Brief Vestibular/Ocular Motor Screening (VOMS). PMC

NW Health Sciences University. (2022). Chiropractic and traumatic brain injuries: Bringing value for TBI and concussion patients. Northwestern Health Sciences University

Patil, S., et al. (2025). Clinical utility of diffusion tensor imaging in sport-related concussion. PMC

Paolini, F., et al. (2025). Diffusion tensor imaging as a neurologic predictor in TBI. MDPI

Richter, S., et al. (2024). Predicting recovery in mild TBI with DTI and biomarkers. The Lancet

Shirley Ryan AbilityLab. (2013). Sensory Organization Test (SOT). Shirley Ryan AbilityLab

Sports Concussion Assessment Tool—SCAT5. (2017). Official SCAT5 form (BJSM). British Journal of Sports Medicine

Tenney, J. R., et al. (2020). American Clinical Neurophysiology Society. Use of quantitative EEG for mTBI—Practice guideline. acns.org

UHC Policy Note. (2024). Computerized dynamic posturography—Medical policy summary. UHC Provider

Zeldovich, M., et al. (2023). Factorial validity of the RPQ across languages (CENTER-TBI). SpringerOpen

Additional clinic/education sources referenced in context

Dr. Alexander Jimenez (clinical perspective & education)

Industry update (biomarkers)